Pathogenesis of chronic pancreatitis: A comprehensive update and a look into the future.
(2009) In Scandinavian Journal of Gastroenterology 44. p.661-663- Abstract
- Chronic pancreatitis is a relatively frequent condition usually caused by alcoholic abuse but also due to recurrent gallstone disease, metabolic endocrine disorders and haemochromatosis, among others. Specific types such as hereditary and autoimmune pancreatitis should be particularly kept in mind and emphasized, as they require specific treatment and attention. The possibility to identify gene mutations has also increased and this is likely to decrease the overall total number of "idiopathic" chronic pancreatitis cases. Pancreatic stellate cells have been identified as potential key players in the progression of chronic pancreatitis and the development of fibrogenesis, which are activated either during repeated attacks of... (More)
- Chronic pancreatitis is a relatively frequent condition usually caused by alcoholic abuse but also due to recurrent gallstone disease, metabolic endocrine disorders and haemochromatosis, among others. Specific types such as hereditary and autoimmune pancreatitis should be particularly kept in mind and emphasized, as they require specific treatment and attention. The possibility to identify gene mutations has also increased and this is likely to decrease the overall total number of "idiopathic" chronic pancreatitis cases. Pancreatic stellate cells have been identified as potential key players in the progression of chronic pancreatitis and the development of fibrogenesis, which are activated either during repeated attacks of necro-inflammation or directly by toxic factors. The inhibition or modulation of pancreatic stellate cells could represent a way of potential intervention in patients with chronic pancreatitis in the future. (Less)
Please use this url to cite or link to this publication:
https://lup.lub.lu.se/record/1302812
- author
- Andersson, Roland LU ; Tingstedt, Bobby LU and Xia, Jinglin
- organization
- publishing date
- 2009
- type
- Contribution to journal
- publication status
- published
- subject
- in
- Scandinavian Journal of Gastroenterology
- volume
- 44
- pages
- 661 - 663
- publisher
- Taylor & Francis
- external identifiers
-
- wos:000266199200002
- pmid:19199163
- scopus:67651148478
- ISSN
- 1502-7708
- DOI
- 10.1080/00365520902718739
- language
- English
- LU publication?
- yes
- id
- 7fc9f4db-6ee7-417c-8dd8-77ca61168dc0 (old id 1302812)
- alternative location
- http://www.ncbi.nlm.nih.gov/pubmed/19199163?dopt=Abstract
- date added to LUP
- 2016-04-04 09:31:16
- date last changed
- 2022-03-31 03:11:37
@article{7fc9f4db-6ee7-417c-8dd8-77ca61168dc0,
abstract = {{Chronic pancreatitis is a relatively frequent condition usually caused by alcoholic abuse but also due to recurrent gallstone disease, metabolic endocrine disorders and haemochromatosis, among others. Specific types such as hereditary and autoimmune pancreatitis should be particularly kept in mind and emphasized, as they require specific treatment and attention. The possibility to identify gene mutations has also increased and this is likely to decrease the overall total number of "idiopathic" chronic pancreatitis cases. Pancreatic stellate cells have been identified as potential key players in the progression of chronic pancreatitis and the development of fibrogenesis, which are activated either during repeated attacks of necro-inflammation or directly by toxic factors. The inhibition or modulation of pancreatic stellate cells could represent a way of potential intervention in patients with chronic pancreatitis in the future.}},
author = {{Andersson, Roland and Tingstedt, Bobby and Xia, Jinglin}},
issn = {{1502-7708}},
language = {{eng}},
pages = {{661--663}},
publisher = {{Taylor & Francis}},
series = {{Scandinavian Journal of Gastroenterology}},
title = {{Pathogenesis of chronic pancreatitis: A comprehensive update and a look into the future.}},
url = {{http://dx.doi.org/10.1080/00365520902718739}},
doi = {{10.1080/00365520902718739}},
volume = {{44}},
year = {{2009}},
}