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- 2001
-
Mark
Hög tid att pröva hypotermi i behandlingen av slaganfall
(
- Contribution to journal › Article
- 2000
-
Mark
The rotating pole test: evaluation of its effectiveness in assessing functional motor deficits following experimental head injury in the rat
(
- Contribution to journal › Article
-
Mark
Subcellular distribution and autophosphorylation of calcium/calmodulin- dependent protein kinase II-α in rat hippocampus in a model of ischemic tolerance
(
- Contribution to journal › Article
-
Mark
Increased survival of embryonic nigral neurons when grafted to hypothermic rats
(
- Contribution to journal › Article
-
Mark
The effect of α-phenyl-tert-butyl nitrone (PBN) on free radical formation in transient focal ischaemia measured by microdialysis and 3,4-dihydroxybenzoate formation
(
- Contribution to journal › Article
-
Mark
Oxidative stress, mitochondrial permeability transition and activation of caspases in calcium ionophore A23187-induced death of cultured striatal neurons
(
- Contribution to journal › Article
- 1999
-
Mark
The tumor suppressor p53 and its response gene p21(WAF1/Cip1) are not markers of neuronal death following transient global cerebral ischemia
(
- Contribution to journal › Article
-
Mark
Cyclosporin A and its nonimmunosuppressive analogue N-Me-Val-4-cyclosporin A mitigate glucose/oxygen deprivation-induced damage to rat cultured hippocampal neurons
(
- Contribution to journal › Article
-
Mark
Rapid decline in protein kinase Cγ levels in the synaptosomal fraction of rat hippocampus after ischemic preconditioning
(
- Contribution to journal › Article
-
Mark
Blockade of the mitochondrial permeability transition pore diminishes infarct size in the rat after transient middle cerebral artery occlusion
(
- Contribution to journal › Article
-
Mark
Changes in protein tyrosine phosphorylation in the rat brain after cerebral ischemia in a model of ischemic tolerance
(
- Contribution to journal › Article
-
Mark
Activation of the extracellular signal-regulated protein kinase cascade in the hippocampal CA1 region in a rat model of global cerebral ischemic preconditioning
(
- Contribution to journal › Article
-
Mark
Differences in the activation of the mitochondrial permeability transition among brain regions in the rat correlate with selective vulnerability
(
- Contribution to journal › Article
-
Mark
Activation of p53 and its target genes p21(WAF1/Cip1) and PAG608/Wig-1 in ischemic preconditioning
(
- Contribution to journal › Article
-
Mark
Acidosis enhances translocation of protein kinase C but not Ca2+/calmodulin-dependent protein kinase II to cell membranes during complete cerebral ischemia
(
- Contribution to journal › Article
-
Mark
The time-course of DNA fragmentation in the choroid plexus and the CA1 region following transient global ischemia in the rat brain. The effect of intra-ischemic hypothermia
(
- Contribution to journal › Article
-
Mark
A simple in vitro model of ischemia based on hippocampal slice cultures and propidium iodide fluorescence
(
- Contribution to journal › Article
-
Mark
Mitochondrial permeability transition induced DNA-fragmentation in the rat hippocampus following hypoglycemia
(
- Contribution to journal › Article
- 1998
-
Mark
Changes in proliferating cell nuclear antigen, a protein involved in DNA repair, in vulnerable hippocampal neurons following global cerebral ischemia
(
- Contribution to journal › Article
-
Mark
The effect of 4β-phorbol-12,13-dibutyrate and staurosporine on the extracellular glutamate levels during ischemia in the rat striatum
(
- Contribution to journal › Article
-
Mark
Cyclosporin A, but not FK 506, protects mitochondria and neurons against hypoglycemic damage and implicates the mitochondrial permeability transition in cell death
(
- Contribution to journal › Article
-
Mark
The effect of hypothermia on the expression of neurotrophin mRNA in the hippocampus following transient cerebral ischemia in the rat
(
- Contribution to journal › Article
-
Mark
Changes in the extracellular levels of glutamate and aspartate during ischemia and hypoglycemia. Effects of hypothermia
(
- Contribution to journal › Article
-
Mark
Novel pharmacologic strategies in the treatment of experimental traumatic brain injury : 1998
(
- Contribution to journal › Scientific review
-
Mark
Regional selective neuronal degeneration after protein phosphatase inhibition in hippocampal slice cultures : Evidence for a MAP kinase-dependent mechanism
(
- Contribution to journal › Article
-
Mark
Sublethal in vitro glucose-oxygen deprivation protects cultured hippocampal neurons against a subsequent severe insult
(
- Contribution to journal › Article
- 1997
-
Mark
High resolution immunogold analysis reveals distinct subcellular compartmentation of protein kinase Cγ and δ in rat Purkinje cells
(
- Contribution to journal › Article
- 1996
-
Mark
Diminished neuronal damage in the rat brain by late treatment with the antipyretic drug dipyrone or cooling following cerebral ischemia
(
- Contribution to journal › Article
-
Mark
Induction of junD mRNA after transient forebrain ischemia in the rat. Effect of hypothermia
(
- Contribution to journal › Article
-
Mark
Increased Levels of Glutamate in Patients with Subarachnoid Haemorrhage as Measured by Intracerebral Microdialysis
(
- Chapter in Book/Report/Conference proceeding › Chapter in report
-
Mark
The immunosuppressant FK506 ameliorates ischaemic damage in the rat brain
(
- Contribution to journal › Article
-
Mark
Intracerebral microdialysis of glutamate and aspartate in two vascular territories after aneurysmal subarachnoid hemorrhage
(
- Contribution to journal › Article
-
Mark
Long-lasting neuroprotective effect of postischemic hypothermia and treatment with an anti-inflammatory/antipyretic drug : Evidence for chronic encephalopathic processes following ischemia
(
- Contribution to journal › Article
-
Mark
Intracellular signal transduction in the postischemic brain.
(
- Contribution to journal › Scientific review
-
Mark
The effect of hypothermia on protein synthesis and the expression of immediate early genes following transient cerebral ischemia.
(
- Contribution to journal › Article
- 1995
-
Mark
Alterations of Ca2+/calmodulin-dependent protein kinase II and its messenger RNA in the rat hippocampus following normo- and hypothermic ischemia
(
- Contribution to journal › Article
-
Mark
Biphasic Expression of the Fos and Jun Families of Transcription Factors Following Transient Forebrain Ischaemia in the Rat. Effect of Hypothermia
(
- Contribution to journal › Article
-
Mark
Persistent Translocation of Ca2+/Calmodulin‐Dependent Protein Kinase II to Synaptic Junctions in the Vulnerable Hippocampal CA1 Region Following Transient Ischemia
(
- Contribution to journal › Article
-
Mark
Persistent Translocation and Inhibition of Ca2+/Calmodulin‐Dependent Protein Kinase II in the Crude Synaptosomal Fraction of the Vulnerable Hippocampus Following Hypoglycemia
(
- Contribution to journal › Article
- 1994
-
Mark
Tyrosine Phosphorylation and Activation of Mitogen‐ Activated Protein Kinase in the Rat Brain Following Transient Cerebral Ischemia
(
- Contribution to journal › Article
-
Mark
Moderate hypothermia mitigates neuronal damage in the rat brain when initiated several hours following transient cerebral ischemia
(
- Contribution to journal › Article
-
Mark
Changes in the Tyrosine Phosphorylation of Mitogen‐Activated Protein Kinase in the Rat Hippocampus During and Following Severe Hypoglycemia
(
- Contribution to journal › Debate/Note/Editorial
- 1993
-
Mark
Postischaemic changes in protein synthesis in the rat brain : effects of hypothermia
(
- Contribution to journal › Article
-
Mark
Heat-shock inhibits protein synthesis and eIF-2 activity in cultured cortical neurons
(
- Contribution to journal › Article
-
Mark
Casein Kinase II Activity in the Postischemic Rat Brain Increases in Brain Regions Resistant to Ischemia and Decreases in Vulnerable Areas
(
- Contribution to journal › Article
-
Mark
Depression of Neuronal Protein Synthesis Initiation by Protein Tyrosine Kinase Inhibitors
(
- Contribution to journal › Article
-
Mark
Initiation of protein synthesis and heat-shock protein-72 expression in the rat brain following severe insulin-induced hypoglycemia
(
- Contribution to journal › Article
-
Mark
Stress-induced inhibition of protein synthesis initiation : Modulation of initiation factor 2 and guanine nucleotide exchange factor activities following transient cerebral ischemia in the rat
(
- Contribution to journal › Article
-
Mark
Ischemia-induced upregulation of excitatory amino acid transport sites
(
- Contribution to journal › Article
-
Mark
Time Course of the Translocation and Inhibition of Protein kinase C During Complete Cerebral Ischemia in the Rat
(
- Contribution to journal › Article
-
Mark
Changes in tyrosine phosphorylation in neocortex following transient cerebral ischaemia
(
- Contribution to journal › Article
-
Mark
Changes in insulin-like growth factor 1 receptor density after transient cerebral ischemia in the rat : Lack of protection against ischemic brain damage following injection of insulin-like growth factor 1
(
- Contribution to journal › Article
-
Mark
Protein phosphorylation and the regulation of mRNA translation following cerebral ischemia
(
- Chapter in Book/Report/Conference proceeding › Book chapter
- 1992
-
Mark
Cerebral protection by AMPA- and NMDA-receptor antagonists administered after severe insulin-induced hypoglycemia
(
- Contribution to journal › Article
-
Mark
NMDA‐receptor blockers but not NBQX, an AMPA‐receptor antagonist, inhibit spreading depression in the rat brain
(
- Contribution to journal › Article
-
Mark
Brain cortical tissue levels of noradrenaline and its glycol metabolites : effects of ischemia and postischemic administration of idazoxan
(
- Contribution to journal › Article
-
Mark
Hypothermia ameliorates neuronal survival when induced 2 hours after ischaemia in the rat
(
- Contribution to journal › Article
-
Mark
Ultrastructural changes in the hippocampal CA1 region following transient cerebral ischemia : evidence against programmed cell death
(
- Contribution to journal › Article
-
Mark
Postischemic blockade of AMPA but not NMDA receptors mitigates neuronal damage in the rat brain following transient severe cerebral ischemia
(
- Contribution to journal › Article
-
Mark
Effects of ischemia on regional ligand binding to adrenoceptors in the rat brain
(
- Contribution to journal › Article
- 1991
-
Mark
Changes in the Activity of Protein Kinase C and the Differential Subcellular Redistribution of Its Isozymes in the Rat Striatum During and Following Transient Forebrain Ischemia
(
- Contribution to journal › Article
-
Mark
Hypothermia Prevents the Ischemia‐Induced Translocation and Inhibition of Protein Kinase C in the Rat Striatum
(
- Contribution to journal › Article
-
Mark
Pathophysiology and pharmacology of cerebral ischemia
(
- Contribution to journal › Article
-
Mark
Lack of protection by the N-methyl-D-asparate receptor blocker dizocilpine (MK-801) after transient severe cerebral ischemia in the rat
(
- Contribution to journal › Article
-
Mark
Changes in pyruvate dehydrogenase complex activity during and following severe insulin-induced hypoglycemia
(
- Contribution to journal › Article
-
Mark
Protein kinases and phosphorylation in neurologic disorders and cell death
(
- Contribution to journal › Scientific review
-
Mark
Extracellular brain cortical levels of noradrenaline in ischemia : - Effects of desipramine and postischemic administration of idazoxan
(
- Contribution to journal › Article
- 1990
-
Mark
Protection against ischemia-induced neuronal damage by the α2-adrenoceptor antagonist idazoxan : influence of time of administration and possible mechanisms of action
(
- Contribution to journal › Article
-
Mark
Protein kinase C is translocated to cell membranes during cerebral ischemia
(
- Contribution to journal › Article
-
Mark
Neuronal damage in the striatum following forebrain ischemia : lack of effect of selective lesions of mesostriatal dopamine neurons
(
- Contribution to journal › Article
-
Mark
Neuronal injury and cerebrovascular disorders
(
- Contribution to journal › Scientific review
-
Mark
Preischemic hyperglycemia and postischemic alteration of rat brain pyruvate dehydrogenase activity
(
- Contribution to journal › Article
-
Mark
Extracellular levels of quinolinic acid are moderately increased in rat neostriatum following severe insulin-induced hypoglycaemia
(
- Contribution to journal › Article
-
Mark
Influence of MK-801 on brain extracellular calcium and potassium activities in severe hypoglycemia
(
- Contribution to journal › Article
-
Mark
Cortical spreading depression is associated with arachidonic acid accumulation and preservation of energy charge
(
- Contribution to journal › Article
- 1989
-
Mark
Postischemic administration of idazoxan, an α-2 adrenergic receptor antagonist, decreases neuronal damage in the rat brain
(
- Contribution to journal › Article
-
Mark
Noradrenaline Metabolism in Neocortex and Hippocampus Following Transient Forebrain Ischemia in Rats : Relation to Development of Selective Neuronal Necrosis
(
- Contribution to journal › Article
-
Mark
Excitatory amino acids and cerebrovascular tone
(
- Contribution to journal › Article
-
Mark
Pyruvate dehydrogenase activity in the rat cerebral cortex following cerebral ischemia
(
- Contribution to journal › Article
-
Mark
Dynamic changes of excitatory amino acid receptors in the rat hippocampus following transient cerebral ischemia
(
- Contribution to journal › Article
-
Mark
Protein kinase C is translocated to the plasma membrane during cerebral ischemia, but is partially inactivated
(
- Chapter in Book/Report/Conference proceeding › Paper in conference proceeding
-
Mark
Mechanisms of glutamate neurotoxicity in cerebral ischemia
(
- Chapter in Book/Report/Conference proceeding › Paper in conference proceeding
-
Mark
Changes in Excitatory Amino Acid Receptor Binding in the Intact and Decorticated Rat Neostriatum Following Insulin‐Induced Hypoglycemia
(
- Contribution to journal › Article
-
Mark
Impairment of protein ubiquitination may cause delayed neuronal death
(
- Contribution to journal › Article
- 1988
-
Mark
The NMDA-antagonist MK-801 reduces extracellular amino acid levels during hypoglycemia and prevents striatal damage
(
- Contribution to journal › Article
- 1987
-
Mark
Excitatory amino acid receptors and ischemic brain damage in the rat
(
- Contribution to journal › Article
-
Mark
Regional differences in arachidonic acid release in rat hippocampal CA1 and CA3 regions during cerebral ischemia
(
- Contribution to journal › Article
-
Mark
Calcium accumulation and neuronal damage in the rat hippocampus following cerebral ischemia
(
- Contribution to journal › Article
-
Mark
Protective effect of lesion to the glutamatergic cortico-striatal projections on the hypoglycemic nerve cell injury in rat striatum
(
- Contribution to journal › Article
- 1986
-
Mark
Effect of Insulin‐Induced Hypoglycemia on the Concentrations of Glutamate and Related Amino Acids and Energy Metabolites in the Intact and Decorticated Rat Neostriatum
(
- Contribution to journal › Article
-
Mark
Circulating catecholamines modulate ischemic brain damage
(
- Contribution to journal › Article
-
Mark
Lesions to the Corticostriatal Pathways Ameliorate Hypoglycemia‐Induced Arachidonic Acid Release
(
- Contribution to journal › Article
-
Mark
Endogenous excitotoxins as possible mediators of ischemic and hypoglycemic brain damage.
(
- Contribution to journal › Article
-
Mark
Flunarizine, a calcium entry blocker, ameliorates ischemic brain damage in the rat
(
- Contribution to journal › Article
-
Mark
The effect of isoflurane on neuronal necrosis following near-complete forebrain ischemia in the rat
(
- Contribution to journal › Article
-
Mark
Chronic dexamethasone pretreatment aggravates ischemic neuronal necrosis
(
- Contribution to journal › Article
-
Mark
Epileptic brain damage : pathophysiology and neurochemical pathology.
(
- Contribution to journal › Scientific review
-
Mark
Cellular and Molecular Events Underlying Epileptic Brain Damage
(
- Contribution to journal › Article
- 1985
-
Mark
Neurochemical Correlates to Selective Neuronal Vulnerability
(
- Chapter in Book/Report/Conference proceeding › Book chapter
-
Mark
Hypoglycemia-induced neuronal damage prevented by an N-methyl-D-aspartate antagonist
(
- Contribution to journal › Article