151 – 200 of 215
- show: 50
- |
- sort: year (new to old)
Close
Embed this list
<iframe src=" "
width=" "
height=" "
allowtransparency="true"
frameborder="0">
</iframe>
- 2012
-
Mark
Risk of venous thromboembolism in first- and second-generation immigrants in Sweden.
(
- Contribution to journal › Article
-
Mark
Risk of haemorrhagic and ischaemic stroke in patients with cancer: A nationwide follow-up study from Sweden.
(
- Contribution to journal › Article
-
Mark
Risk of pulmonary embolism in patients with autoimmune disorders: a nationwide follow-up study from Sweden.
(
- Contribution to journal › Article
-
Mark
Socioeconomic and occupational risk factors for venous thromboembolism in Sweden: A nationwide epidemiological study.
(
- Contribution to journal › Article
-
Mark
Occupational Risk Factors for Systemic Lupus Erythematosus: a Nationwide Study Based on Hospitalizations in Sweden.
(
- Contribution to journal › Article
-
Mark
Risk of coronary heart disease in patients with cancer: A nationwide follow-up study from Sweden.
(
- Contribution to journal › Article
-
Mark
Family history and risk of hospital treatment for varicose veins in Sweden.
(
- Contribution to journal › Article
-
Mark
A nationwide family study of pulmonary embolism: Identification of high risk families with increased risk of hospitalized and fatal pulmonary embolism.
(
- Contribution to journal › Article
-
Mark
Neighborhood deprivation and hospitalization for venous thromboembolism in Sweden.
(
- Contribution to journal › Article
-
Mark
Multiplex sibling history of coronary heart disease is a strong risk factor for coronary heart disease.
(
- Contribution to journal › Article
-
Mark
Shared familial aggregation of susceptibility to different manifestations of venous thromboembolism: a nationwide family study in Sweden.
(
- Contribution to journal › Letter
-
Mark
Autoimmune diseases and venous thromboembolism : a review of the literature
(
- Contribution to journal › Article
-
Mark
Stark ärftlighet för varicer bland svenskar, visar nationell studie
(
- Contribution to journal › Debate/Note/Editorial
- 2011
-
Mark
Age- and gender-specific familial risks for venous thromboembolism: a nationwide epidemiological study based on hospitalizations in sweden.
(
- Contribution to journal › Article
-
Mark
Venous Thromboembolism Does Not Share Strong Familial Susceptibility with Ischemic Stroke: A Nationwide Family Study in Sweden.
(
- Contribution to journal › Article
-
Mark
Parental history and venous thromboembolism: a nationwide study of age and sex-specific familial risks in Sweden.
(
- Contribution to journal › Article
-
Mark
Familial risks of unusual forms of venous thrombosis: a nationwide epidemiological study in Sweden.
(
- Contribution to journal › Article
-
Mark
Venous thromboembolism does not share strong familial susceptibility with coronary heart disease: a nationwide family study in Sweden.
(
- Contribution to journal › Article
-
Mark
Familial risk factors shared by venous thromboembolism and cancer: A nationwide epidemiological study of Swedish families.
(
- Contribution to journal › Article
-
Mark
Determination of age-specific and sex-specific familial risks for the different manifestations of venous thromboembolism: A nationwide family study in Sweden.
(
- Contribution to journal › Article
- 2005
-
Mark
Large deletions of the PROS1 gene in a large fraction of mutationnegative patients with protein S deficiency
(
- Contribution to journal › Article
- 2004
-
Mark
Co-segregation of the PROS1 locus and protein S deficiency in families having no detectable mutations in PROS1.
(
- Contribution to journal › Article
- 2002
-
Mark
Naturally occurring mutations in the thrombomodulin gene leading to impaired expression and function
(
- Contribution to journal › Article
-
Mark
Genetic and phenotypic variability between families with hereditary protein S deficiency
(
- Contribution to journal › Article
- 2001
-
Mark
Improved hemoglobin status and reduced menstrual blood loss among female carriers of factor V Leiden--an evolutionary advantage?
(
- Contribution to journal › Letter
- 1999
-
Mark
Factor V Q506 (resistance to activated protein C) and prognosis after acute coronary syndrome
(
- Contribution to journal › Article
-
Mark
Thrombophilia as a multigenic disease
(
- Contribution to journal › Article
-
Mark
The Ala25-Thr mutation in the thrombomodulin gene is not frequent in Swedish patients suffering from ischemic heart disease [9]
(
- Contribution to journal › Letter
- 1998
-
Mark
A common 4G allele in the promoter of the plasminogen activator inhibitor-1 (PAI-1) gene as a risk factor for pulmonary embolism and arterial thrombosis in hereditary protein S deficiency
(
- Contribution to journal › Article
-
Mark
A new direct, fast and quantitative enzyme-linked ligandsorbent assay for measurement of free protein S antigen
(
- Contribution to journal › Article
-
Mark
Clarification of the risk for venous thrombosis associated with hereditary protein S deficiency by investigation of a large kindred with a characterized gene defect
(
- Contribution to journal › Article
-
Mark
The A20210 allele of the prothrombin gene is frequently associated with the factor V Arg 506 to Gln mutation but not with protein S deficiency in thrombophilic families
(
- Contribution to journal › Letter
-
Mark
The A20210 allele of the prothrombin gene is frequently associated with the factor V Arg 506 to Gln mutation but not with protein S deficiency in thrombophilic families [3]
(
- Contribution to journal › Letter
- 1997
-
Mark
A novel thrombomodulin gene mutation in a patient suffering from sagittal sinus thrombosis
(
- Contribution to journal › Article
-
Mark
The 20210 A allele of the prothrombin gene is a common risk factor among Swedish outpatients with verified deep venous thrombosis
(
- Contribution to journal › Debate/Note/Editorial
-
Mark
Genetic and phenotypic analysis of a large (122-member) protein S-deficient kindred provides an explanation for the familial coexistence of type I and type III plasma phenotypes
(
- Contribution to journal › Article
-
Mark
Activated protein C resistance caused by a common factor V mutation has a single origin
(
- Contribution to journal › Article
-
Mark
The factor VR506Q mutation causing APC resistance is highly prevalent amongst unselected outpatients with clinically suspected deep venous thrombosis
(
- Contribution to journal › Article
-
Mark
Evaluation of original and modified APC-resistance tests in unselected outpatients with clinically suspected thrombosis and in healthy controls
(
- Contribution to journal › Article
-
Mark
A common thrombomodulin amino acid dimorphism is associated with myocardial infarction
(
- Contribution to journal › Article
-
Mark
Activated protein C resistance due to a common factor V gene mutation is a major risk factor for venous thrombosis
(
- Contribution to journal › Article
-
Mark
The factor VR5O6Q mutation causing APC resistance is highly prevalent amongst unselected outpatients with clinically suspected deep venous thrombosis
(
- Contribution to journal › Article
-
Mark
Activated protein C resistance : Clinical implications
(
- Contribution to journal › Scientific review
- 1996
-
Mark
Familial thrombophilia : clinical and molecular analysis of Swedish families with inherited resistance to activated protein C or protein S deficiency
1996) In Scandinavian journal of clinical and laboratory investigation. Supplementum 56(226). p.19-46(
- Contribution to journal › Scientific review
-
Mark
Elevated levels of prothrombin activation fragment 1 + 2 in plasma from patients with heterozygous Arg506 to Gln mutation in the factor V gene (APC-resistance) and/or inherited protein S deficiency
(
- Contribution to journal › Article
-
Mark
Resistance to activated protein C due to a factor V gene mutation : The most common inherited risk factor of thrombosis
(
- Contribution to journal › Article
-
Mark
Prevalence of factor V gene mutation amongst myocardial infarction patients and healthy controls is higher in Sweden than in other countries
(
- Contribution to journal › Article
-
Mark
Resistance to activated protein C, the FV:Q506 allele, and venous thrombosis
(
- Contribution to journal › Scientific review
-
Mark
High prevalence of the FVR506Q mutation causing APC resistance in a region of southern Sweden with a high incidence of venous thrombosis.
(
- Contribution to journal › Letter
-
Mark
Inherited resistance to activated protein C caused by presence of the FV:Q506 allele as a basis of venous thrombosis
(
- Contribution to journal › Article