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- 2024
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Mark
Intracellular C3 protects β-cells from IL-1β-driven cytotoxicity via interaction with Fyn-related kinase
(
- Contribution to journal › Article
- 2022
-
Mark
Alternative splicing encodes functional intracellular CD59 isoforms that mediate insulin secretion and are down-regulated in diabetic islets
(
- Contribution to journal › Article
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Mark
The structure of insulin granule core determines secretory capacity being reduced in type-2 diabetes
2022)(
- Working paper/Preprint › Preprint in preprint archive
- 2021
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Mark
Complement in metabolic disease : metaflammation and a two-edged sword
(
- Contribution to journal › Scientific review
- 2019
-
Mark
Intracellular cytosolic complement component C3 regulates cytoprotective autophagy in pancreatic beta cells by interaction with ATG16L1
(
- Contribution to journal › Debate/Note/Editorial
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Mark
Complement Component C3 Is Highly Expressed in Human Pancreatic Islets and Prevents β Cell Death via ATG16L1 Interaction and Autophagy Regulation
(
- Contribution to journal › Article
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Mark
A cryptic non-GPI-anchored cytosolic isoform of CD59 controls insulin exocytosis in pancreatic β-cells by interaction with SNARE proteins
2019) In FASEB journal : official publication of the Federation of American Societies for Experimental Biology 33(11). p.12425-12434(
- Contribution to journal › Article
- 2017
-
Mark
Non-traditional roles of complement in type 2 diabetes : Metabolism, insulin secretion and homeostasis
(
- Contribution to journal › Article
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Mark
Complement C3 Associates With Incidence of Diabetes, but No Evidence of a Causal Relationship
(
- Contribution to journal › Article
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Mark
The human serum protein C4b-binding protein inhibits pancreatic IAPP-induced inflammasome activation
(
- Contribution to journal › Article