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- 2019
- Maintenance of near normal bone mass and architecture in lethally irradiated female mice following adoptive transfer with as few as 750 purified hematopoietic stem cells (
- 2018
- The Impact of Amyloid Peptides on Pericytes in Alzheimer's Disease (
- Mitophagy : A Mechanism for Plant Growth and Survival (
- 2017
- HAMLET – A protein-lipid complex with broad tumoricidal activity (
- Smac and Smac mimetics in breast cancer cell death (
- Ca2+ involvement in activation of extracellular-signalregulated- kinase 1/2 and m-calpain after axotomy of the sciatic nerve (
- 2016
- Cellular targets of HAMLET, their role in tumor cell death and therapeutic potential (
- Global binding pattern of the Wilms' tumor gene 1 (WT1) +17AA -KTS isoform in leukemic cells (
- 2015
- Sensitive detection of lysosomal membrane permeabilization by lysosomal galectin puncta assay (
- 2014
- Extracellular hemoglobin - mediator of inflammation and cell death in the choroid plexus following preterm intraventricular hemorrhage (
- Novel In Situ Activity Assays for the Quantitative Molecular Analysis of Neurodegenerative Processes in the Retina (
- The mitochondrial outer membrane AAA ATPase AtOM66 affects cell death and pathogen resistance in Arabidopsis thaliana (
- IL-17 protects T cells from apoptosis and contributes to development of ALPS-like phenotypes (
- 2013
- Roles of protein kinase C in cell death and breast cancer (
- Endothelial Cell Death and Intimal Foam Cell Accumulation in the Coronary Artery of Infected Hypercholesterolemic Minipigs (
- 2012
- The mechanism of HAMLET-induced cell death - cellular signalling, oncogenes and clinical perspectives (
- Identification of a novel protein kinase Cδ-Smac complex that dissociates during paclitaxel-induced cell death. (
- A novel initiation mechanism of death in Streptococcus pneumoniae induced by the human milk protein-lipid complex HAMLET and activated during physiological death (
- Oleic acid is a key cytotoxic component of HAMLET-like complexes (
- 2011
- Monomeric C-reactive protein modulates classic complement activation on necrotic cells (