Christian Lood (Former)
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- 2012
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Mark
Increased C1q, C4 and C3 deposition on platelets in patients with systemic lupus erythematosus - a possible link to venous thrombosis?
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- Contribution to journal › Article
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Mark
IgG glycan hydrolysis by endoglycosidase S diminishes the proinflammatory properties of immune complexes from patients with systemic lupus erythematosus: A possible new treatment?
(
- Contribution to journal › Article
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Mark
Vaccination against encapsulated bacteria in hereditary C2 deficiency results in antibody response and opsonization due to antibody-dependent complement activation.
(
- Contribution to journal › Article
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Mark
Neutrophil Extracellular Traps That Are Not Degraded in Systemic Lupus Erythematosus Activate Complement Exacerbating the Disease.
(
- Contribution to journal › Article
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Mark
IgG glycan hydrolysis by EndoS diminishes the pro-inflammatory properties of immune complexes from patients with SLE - a possible new treatment?
(
- Contribution to journal › Published meeting abstract
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Mark
Combination of Autoantibodies Against Different Histone Proteins Influences Complement-dependent Phagocytosis of Necrotic Cell Material by Polymorphonuclear Leukocytes in Systemic Lupus Erythematosus.
(
- Contribution to journal › Article
- 2011
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Mark
Protein synthesis of the pro-inflammatory S100A8/A9 complex in plasmacytoid dendritic cells and cell surface S100A8/A9 on leukocyte subpopulations in systemic lupus erythematosus
(
- Contribution to journal › Article
- 2010
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Mark
Recombinant human complement component C2 produced in a human cell line restores the classical complement pathway activity in-vitro: an alternative treatment for C2 deficiency diseases
(
- Contribution to journal › Article
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Mark
The IgG specific endoglycosidase EndoS inhibits both cellular and complement mediated autoimmune hemolysis.
(
- Contribution to journal › Article
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Mark
Platelet transcriptional profile and protein expression in patients with systemic lupus erythematosus: up-regulation of the type I interferon system is strongly associated with vascular disease.
(
- Contribution to journal › Article