Maternal APOE ε2 as a possible risk factor for elevated prenatal Pb levels
(2024) In Environmental Research 260. p.1-12- Abstract
Lead (Pb) is a global contaminant associated with multiple adverse health effects. Humans are especially vulnerable during critical developmental stages. During pregnancy, exposure to Pb can occur through diet and release from maternal bones. Apolipoprotein E gene (APOE) variants (ɛ2, ɛ3, ɛ4 alleles) may influence sex steroid hormones, bone metabolism, and Pb kinetics. We examined the interplay among maternal APOE (mAPOE) genotypes, fetal sex, parity, and Pb in maternal and cord blood (mB-Pb, CB-Pb) using linear regression models. Our study involved 817 pregnant women and 772 newborns with measured adequate levels of zinc and selenium. We compared carriers of the ε2 and ε4 alleles to those with the ε3/ε3 genotype. The geometric means... (More)
Lead (Pb) is a global contaminant associated with multiple adverse health effects. Humans are especially vulnerable during critical developmental stages. During pregnancy, exposure to Pb can occur through diet and release from maternal bones. Apolipoprotein E gene (APOE) variants (ɛ2, ɛ3, ɛ4 alleles) may influence sex steroid hormones, bone metabolism, and Pb kinetics. We examined the interplay among maternal APOE (mAPOE) genotypes, fetal sex, parity, and Pb in maternal and cord blood (mB-Pb, CB-Pb) using linear regression models. Our study involved 817 pregnant women and 772 newborns with measured adequate levels of zinc and selenium. We compared carriers of the ε2 and ε4 alleles to those with the ε3/ε3 genotype. The geometric means (range) of mB-Pb and CB-Pb were 11.1 (3.58-87.6) and 9.31 (1.82-47.0) ng/g, respectively. In cases with female fetuses, the maternal mAPOE ε2 allele was associated with higher, while the mAPOE ε4 allele was associated with lower mB-Pb and CB-Pb levels. Nulliparity increased the strength of the observed associations. These findings highlight the significance of mAPOE genetics, fetal sex, and parity in prenatal Pb kinetics. Notably, the maternal ε2 allele may increase the risk of Pb exposure.
(Less)
- author
- publishing date
- 2024-11-01
- type
- Contribution to journal
- publication status
- published
- keywords
- Adult, Female, Humans, Infant, Newborn, Male, Pregnancy, Young Adult, Apolipoprotein E2/genetics, Environmental Pollutants/blood, Fetal Blood/chemistry, Genotype, Lead/blood, Maternal Exposure/adverse effects, Risk Factors
- in
- Environmental Research
- volume
- 260
- article number
- 119583
- pages
- 1 - 12
- publisher
- Elsevier
- external identifiers
-
- scopus:85198532100
- pmid:38992759
- ISSN
- 1096-0953
- DOI
- 10.1016/j.envres.2024.119583
- language
- English
- LU publication?
- no
- additional info
- Copyright © 2024 The Authors. Published by Elsevier Inc. All rights reserved.
- id
- 036cb7d6-c0aa-498c-9190-a06e96f0d7ce
- date added to LUP
- 2025-02-18 17:05:42
- date last changed
- 2025-06-11 12:59:33
@article{036cb7d6-c0aa-498c-9190-a06e96f0d7ce, abstract = {{<p>Lead (Pb) is a global contaminant associated with multiple adverse health effects. Humans are especially vulnerable during critical developmental stages. During pregnancy, exposure to Pb can occur through diet and release from maternal bones. Apolipoprotein E gene (APOE) variants (ɛ2, ɛ3, ɛ4 alleles) may influence sex steroid hormones, bone metabolism, and Pb kinetics. We examined the interplay among maternal APOE (mAPOE) genotypes, fetal sex, parity, and Pb in maternal and cord blood (mB-Pb, CB-Pb) using linear regression models. Our study involved 817 pregnant women and 772 newborns with measured adequate levels of zinc and selenium. We compared carriers of the ε2 and ε4 alleles to those with the ε3/ε3 genotype. The geometric means (range) of mB-Pb and CB-Pb were 11.1 (3.58-87.6) and 9.31 (1.82-47.0) ng/g, respectively. In cases with female fetuses, the maternal mAPOE ε2 allele was associated with higher, while the mAPOE ε4 allele was associated with lower mB-Pb and CB-Pb levels. Nulliparity increased the strength of the observed associations. These findings highlight the significance of mAPOE genetics, fetal sex, and parity in prenatal Pb kinetics. Notably, the maternal ε2 allele may increase the risk of Pb exposure.</p>}}, author = {{Palir, Neza and Stajnko, Anja and Mazej, Darja and France Štiglic, Alenka and Rosolen, Valentina and Mariuz, Marika and Ronfani, Luca and Snoj Tratnik, Janja and Runkel, Agneta Annika and Tursunova, Veronika and Marc, Janja and Prpić, Igor and Špirić, Zdravko and Barbone, Fabio and Horvat, Milena and Falnoga, Ingrid}}, issn = {{1096-0953}}, keywords = {{Adult; Female; Humans; Infant, Newborn; Male; Pregnancy; Young Adult; Apolipoprotein E2/genetics; Environmental Pollutants/blood; Fetal Blood/chemistry; Genotype; Lead/blood; Maternal Exposure/adverse effects; Risk Factors}}, language = {{eng}}, month = {{11}}, pages = {{1--12}}, publisher = {{Elsevier}}, series = {{Environmental Research}}, title = {{Maternal APOE ε2 as a possible risk factor for elevated prenatal Pb levels}}, url = {{http://dx.doi.org/10.1016/j.envres.2024.119583}}, doi = {{10.1016/j.envres.2024.119583}}, volume = {{260}}, year = {{2024}}, }