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Resialylation of sialic acid deficit vascular endothelium, circulating cells and macromolecules may counteract the development of atherosclerosis: A hypothesis.

Lindberg, Gunnar LU (2007) In Atherosclerosis 192(2). p.243-245
Abstract
Deficit sialylation of vascular endothelium, circulating cells and macromolecules has been associated with the development of atherosclerosis. On the other hand, an elevated serum level of sialic acid is a long-lasting marker of atherosclerosis and complications from atherosclerosis. One may speculate that the inverse associations with atherosclerosis risk are due to some common underlying cause. One mission for the elevated serum sialic acid level might be to act as a substrate for resialylation of sialic acid deficit structures and thereby counteract the atherosclerotic process.
Please use this url to cite or link to this publication:
author
organization
publishing date
type
Contribution to journal
publication status
published
subject
keywords
sialic acid, resialylation, atherosclerosis, hypothesis
in
Atherosclerosis
volume
192
issue
2
pages
243 - 245
publisher
Elsevier
external identifiers
  • wos:000247225900002
  • scopus:34248170681
ISSN
1879-1484
DOI
10.1016/j.atherosclerosis.2007.03.011
language
English
LU publication?
yes
id
fb28f2c6-8207-45c2-9aed-22b59b4cb79b (old id 167744)
alternative location
http://www.ncbi.nlm.nih.gov/entrez/query.fcgi?cmd=Retrieve&db=PubMed&list_uids=17420020&dopt=Abstract
date added to LUP
2007-07-25 14:18:49
date last changed
2017-01-29 03:27:45
@article{fb28f2c6-8207-45c2-9aed-22b59b4cb79b,
  abstract     = {Deficit sialylation of vascular endothelium, circulating cells and macromolecules has been associated with the development of atherosclerosis. On the other hand, an elevated serum level of sialic acid is a long-lasting marker of atherosclerosis and complications from atherosclerosis. One may speculate that the inverse associations with atherosclerosis risk are due to some common underlying cause. One mission for the elevated serum sialic acid level might be to act as a substrate for resialylation of sialic acid deficit structures and thereby counteract the atherosclerotic process.},
  author       = {Lindberg, Gunnar},
  issn         = {1879-1484},
  keyword      = {sialic acid,resialylation,atherosclerosis,hypothesis},
  language     = {eng},
  number       = {2},
  pages        = {243--245},
  publisher    = {Elsevier},
  series       = {Atherosclerosis},
  title        = {Resialylation of sialic acid deficit vascular endothelium, circulating cells and macromolecules may counteract the development of atherosclerosis: A hypothesis.},
  url          = {http://dx.doi.org/10.1016/j.atherosclerosis.2007.03.011},
  volume       = {192},
  year         = {2007},
}