microRNA-125 distinguishes developmentally generated and adult-born olfactory bulb interneurons.
(2014) In Development: For advances in developmental biology and stem cells 141(7). p.1580-1588- Abstract
- New neurons, originating from the subventricular zone, are continuously integrating into neuronal circuitry in the olfactory bulb (OB). Using a transgenic sensor mouse, we found that adult-born OB interneurons express microRNA-125 (miR-125), whereas the pre-existing developmentally generated OB interneurons represent a unique population of cells in the adult brain, without miR-125 activity. Stable inhibition of miR-125 in newborn OB neurons resulted in enhanced dendritic morphogenesis, as well as in increased synaptic activation in response to odour sensory stimuli. These data demonstrate that miR-125 controls functional synaptic integration of adult-born OB interneurons. Our results also suggest that absence of an otherwise broadly... (More)
- New neurons, originating from the subventricular zone, are continuously integrating into neuronal circuitry in the olfactory bulb (OB). Using a transgenic sensor mouse, we found that adult-born OB interneurons express microRNA-125 (miR-125), whereas the pre-existing developmentally generated OB interneurons represent a unique population of cells in the adult brain, without miR-125 activity. Stable inhibition of miR-125 in newborn OB neurons resulted in enhanced dendritic morphogenesis, as well as in increased synaptic activation in response to odour sensory stimuli. These data demonstrate that miR-125 controls functional synaptic integration of adult-born OB interneurons. Our results also suggest that absence of an otherwise broadly expressed miRNA is a novel mechanism with which to achieve neuronal subtype specification. (Less)
Please use this url to cite or link to this publication:
https://lup.lub.lu.se/record/4383839
- author
- Åkerblom, Malin LU ; Petri, Rebecca LU ; Sachdeva, Rohit LU ; Klüssendorf, Thies LU ; Mattsson, Bengt LU ; Gentner, Bernhard and Jakobsson, Johan LU
- organization
- publishing date
- 2014
- type
- Contribution to journal
- publication status
- published
- subject
- in
- Development: For advances in developmental biology and stem cells
- volume
- 141
- issue
- 7
- pages
- 1580 - 1588
- publisher
- The Company of Biologists Ltd
- external identifiers
-
- pmid:24598163
- wos:000333184500017
- scopus:84896271384
- pmid:24598163
- ISSN
- 1477-9129
- DOI
- 10.1242/dev.101659
- language
- English
- LU publication?
- yes
- id
- 3b287be7-9679-4160-a5bd-4fc0ce200cc7 (old id 4383839)
- alternative location
- http://www.ncbi.nlm.nih.gov/pubmed/24598163?dopt=Abstract
- date added to LUP
- 2016-04-01 10:06:05
- date last changed
- 2024-02-21 08:03:27
@article{3b287be7-9679-4160-a5bd-4fc0ce200cc7, abstract = {{New neurons, originating from the subventricular zone, are continuously integrating into neuronal circuitry in the olfactory bulb (OB). Using a transgenic sensor mouse, we found that adult-born OB interneurons express microRNA-125 (miR-125), whereas the pre-existing developmentally generated OB interneurons represent a unique population of cells in the adult brain, without miR-125 activity. Stable inhibition of miR-125 in newborn OB neurons resulted in enhanced dendritic morphogenesis, as well as in increased synaptic activation in response to odour sensory stimuli. These data demonstrate that miR-125 controls functional synaptic integration of adult-born OB interneurons. Our results also suggest that absence of an otherwise broadly expressed miRNA is a novel mechanism with which to achieve neuronal subtype specification.}}, author = {{Åkerblom, Malin and Petri, Rebecca and Sachdeva, Rohit and Klüssendorf, Thies and Mattsson, Bengt and Gentner, Bernhard and Jakobsson, Johan}}, issn = {{1477-9129}}, language = {{eng}}, number = {{7}}, pages = {{1580--1588}}, publisher = {{The Company of Biologists Ltd}}, series = {{Development: For advances in developmental biology and stem cells}}, title = {{microRNA-125 distinguishes developmentally generated and adult-born olfactory bulb interneurons.}}, url = {{http://dx.doi.org/10.1242/dev.101659}}, doi = {{10.1242/dev.101659}}, volume = {{141}}, year = {{2014}}, }