Myeloid Kdm6b deficiency results in advanced atherosclerosis
(2018) In Atherosclerosis 275. p.156-165- Abstract
BACKGROUND AND AIMS: Atherosclerosis is a lipid-driven chronic inflammatory disorder of the arteries, and monocytes and macrophages play a central role in this process. Within the atherosclerotic lesion, macrophages can scavenge modified lipids and become the so-called foam cells. We previously reported that the epigenetic enzyme Kdm6b (also known as Jmjd3) controls the pro-fibrotic transcriptional profile of peritoneal foam cells. Given the importance of these cells in atherosclerosis, we now studied the effect of myeloid Kdm6b on disease progression.
METHODS: Bone marrow of myeloid Kdm6b deficient (Kdm6bdel) mice or wild type littermates (Kdm6bwt) was transplanted to lethally irradiated Ldlr-/- mice fed a high fat diet for 9... (More)
BACKGROUND AND AIMS: Atherosclerosis is a lipid-driven chronic inflammatory disorder of the arteries, and monocytes and macrophages play a central role in this process. Within the atherosclerotic lesion, macrophages can scavenge modified lipids and become the so-called foam cells. We previously reported that the epigenetic enzyme Kdm6b (also known as Jmjd3) controls the pro-fibrotic transcriptional profile of peritoneal foam cells. Given the importance of these cells in atherosclerosis, we now studied the effect of myeloid Kdm6b on disease progression.
METHODS: Bone marrow of myeloid Kdm6b deficient (Kdm6bdel) mice or wild type littermates (Kdm6bwt) was transplanted to lethally irradiated Ldlr-/- mice fed a high fat diet for 9 weeks to induce atherosclerosis.
RESULTS: Lesion size was similar in Kdm6bwt and Kdm6bdel transplanted mice. However, lesions of Kdm6bdel mice contained more collagen and were more necrotic. Pathway analysis on peritoneal foam cells showed that the pathway involved in leukocyte chemotaxis was most significantly upregulated. Although macrophage and neutrophil content was similar after 9 weeks of high fat diet feeding, the relative increase in collagen content and necrosis revealed that atherosclerotic lesions in Kdm6bdel mice progress faster.
CONCLUSION: Myeloid Kdm6b deficiency results in more advanced atherosclerosis.
(Less)
- author
- publishing date
- 2018-08
- type
- Contribution to journal
- publication status
- published
- subject
- keywords
- Animals, Aorta/enzymology, Aortic Diseases/enzymology, Atherosclerosis/enzymology, Cells, Cultured, Chemotaxis, Leukocyte, Collagen/metabolism, Diet, High-Fat, Disease Models, Animal, Disease Progression, Female, Fibrosis, Foam Cells/enzymology, Jumonji Domain-Containing Histone Demethylases/deficiency, Macrophages, Peritoneal/enzymology, Mice, Inbred C57BL, Mice, Knockout, Necrosis, Neutrophil Infiltration, Plaque, Atherosclerotic, Receptors, LDL/deficiency, Time Factors
- in
- Atherosclerosis
- volume
- 275
- pages
- 156 - 165
- publisher
- Elsevier
- external identifiers
-
- scopus:85048328199
- pmid:29908485
- ISSN
- 1879-1484
- DOI
- 10.1016/j.atherosclerosis.2018.05.052
- language
- English
- LU publication?
- no
- additional info
- Copyright © 2018 The Authors. Published by Elsevier B.V. All rights reserved.
- id
- 8e5d9889-b724-4f11-bca2-0ea9320f3a23
- date added to LUP
- 2025-01-22 15:23:44
- date last changed
- 2025-04-17 10:36:49
@article{8e5d9889-b724-4f11-bca2-0ea9320f3a23, abstract = {{<p>BACKGROUND AND AIMS: Atherosclerosis is a lipid-driven chronic inflammatory disorder of the arteries, and monocytes and macrophages play a central role in this process. Within the atherosclerotic lesion, macrophages can scavenge modified lipids and become the so-called foam cells. We previously reported that the epigenetic enzyme Kdm6b (also known as Jmjd3) controls the pro-fibrotic transcriptional profile of peritoneal foam cells. Given the importance of these cells in atherosclerosis, we now studied the effect of myeloid Kdm6b on disease progression.</p><p>METHODS: Bone marrow of myeloid Kdm6b deficient (Kdm6bdel) mice or wild type littermates (Kdm6bwt) was transplanted to lethally irradiated Ldlr-/- mice fed a high fat diet for 9 weeks to induce atherosclerosis.</p><p>RESULTS: Lesion size was similar in Kdm6bwt and Kdm6bdel transplanted mice. However, lesions of Kdm6bdel mice contained more collagen and were more necrotic. Pathway analysis on peritoneal foam cells showed that the pathway involved in leukocyte chemotaxis was most significantly upregulated. Although macrophage and neutrophil content was similar after 9 weeks of high fat diet feeding, the relative increase in collagen content and necrosis revealed that atherosclerotic lesions in Kdm6bdel mice progress faster.</p><p>CONCLUSION: Myeloid Kdm6b deficiency results in more advanced atherosclerosis.</p>}}, author = {{Neele, Annette E and Gijbels, Marion J J and van der Velden, Saskia and Hoeksema, Marten A and Boshuizen, Marieke C S and Prange, Koen H M and Chen, Hung-Jen and Van den Bossche, Jan and van Roomen, Cindy P P A and Shami, Annelie and Levels, Johannes H M and Kroon, Jeffrey and Lucas, Tina and Dimmeler, Stefanie and Lutgens, Esther and de Winther, Menno P J}}, issn = {{1879-1484}}, keywords = {{Animals; Aorta/enzymology; Aortic Diseases/enzymology; Atherosclerosis/enzymology; Cells, Cultured; Chemotaxis, Leukocyte; Collagen/metabolism; Diet, High-Fat; Disease Models, Animal; Disease Progression; Female; Fibrosis; Foam Cells/enzymology; Jumonji Domain-Containing Histone Demethylases/deficiency; Macrophages, Peritoneal/enzymology; Mice, Inbred C57BL; Mice, Knockout; Necrosis; Neutrophil Infiltration; Plaque, Atherosclerotic; Receptors, LDL/deficiency; Time Factors}}, language = {{eng}}, pages = {{156--165}}, publisher = {{Elsevier}}, series = {{Atherosclerosis}}, title = {{Myeloid Kdm6b deficiency results in advanced atherosclerosis}}, url = {{http://dx.doi.org/10.1016/j.atherosclerosis.2018.05.052}}, doi = {{10.1016/j.atherosclerosis.2018.05.052}}, volume = {{275}}, year = {{2018}}, }