Skip to main content

Lund University Publications

LUND UNIVERSITY LIBRARIES

Herpes Simplex Virus Hepatitis : An Overlooked Cause of Acute Liver Failure Across a Broad Spectrum of Immunocompromise

Mondelli, Mario U. ; Ludovisi, Serena ; Di Matteo, Angela ; Şahin, Gülşen Özkaya LU and Irving, William (2026) In Liver International 46(7).
Abstract

Background and Aims: Herpes simplex virus (HSV) infection is common worldwide, but hepatic involvement is rare. HSV hepatitis is an uncommon yet frequently catastrophic cause of acute hepatitis and acute liver failure (ALF), accounting for < 1% of ALF cases while carrying very high mortality when diagnosis and antiviral therapy are delayed. Although classically associated with profound immunosuppression and late pregnancy, HSV hepatitis is increasingly recognized in patients with less obvious or “non-traditional” forms of immune dysfunction. Methods: We describe three cases of HSV hepatitis presenting with distinct risk profiles and clinical manifestations and review the diagnostic and therapeutic challenges associated with this... (More)

Background and Aims: Herpes simplex virus (HSV) infection is common worldwide, but hepatic involvement is rare. HSV hepatitis is an uncommon yet frequently catastrophic cause of acute hepatitis and acute liver failure (ALF), accounting for < 1% of ALF cases while carrying very high mortality when diagnosis and antiviral therapy are delayed. Although classically associated with profound immunosuppression and late pregnancy, HSV hepatitis is increasingly recognized in patients with less obvious or “non-traditional” forms of immune dysfunction. Methods: We describe three cases of HSV hepatitis presenting with distinct risk profiles and clinical manifestations and review the diagnostic and therapeutic challenges associated with this condition. Results: Clinical presentation was nonspecific in all cases, and mucocutaneous lesions were absent or delayed, contributing to diagnostic uncertainty. All patients exhibited marked aminotransferase elevation with relatively modest hyperbilirubinemia (“anicteric hepatitis”), frequently accompanied by cytopenias and coagulopathy. Delayed recognition was associated with rapid clinical deterioration, whereas earlier initiation of intravenous acyclovir was associated with biochemical and clinical improvement. Diagnosis was established using polymerase chain reaction–based detection of HSV DNA in blood and tissue samples. Conclusions: HSV hepatitis remains an under-recognized cause of ALF across a broad spectrum of immunocompromised and seemingly immunocompetent patients. Given the narrow therapeutic window and favourable safety profile of acyclovir, empiric intravenous antiviral therapy should be considered in patients with ALF of indeterminate aetiology while diagnostic testing is pending, particularly in high-risk clinical settings. Increased awareness of this overlooked diagnosis may improve outcomes and prevent avoidable mortality.

(Less)
Please use this url to cite or link to this publication:
author
; ; ; and
author collaboration
organization
publishing date
type
Contribution to journal
publication status
published
subject
keywords
acute liver failure, acyclovir, hepatitis, herpes simplex virus
in
Liver International
volume
46
issue
7
article number
e70756
publisher
Wiley-Blackwell
external identifiers
  • pmid:42324722
  • scopus:105042617067
ISSN
1478-3223
DOI
10.1111/liv.70756
language
English
LU publication?
yes
id
a28f8f3c-ea30-4a46-a8e2-3de3e416c30c
date added to LUP
2026-07-03 14:57:01
date last changed
2026-07-04 03:00:04
@article{a28f8f3c-ea30-4a46-a8e2-3de3e416c30c,
  abstract     = {{<p>Background and Aims: Herpes simplex virus (HSV) infection is common worldwide, but hepatic involvement is rare. HSV hepatitis is an uncommon yet frequently catastrophic cause of acute hepatitis and acute liver failure (ALF), accounting for &lt; 1% of ALF cases while carrying very high mortality when diagnosis and antiviral therapy are delayed. Although classically associated with profound immunosuppression and late pregnancy, HSV hepatitis is increasingly recognized in patients with less obvious or “non-traditional” forms of immune dysfunction. Methods: We describe three cases of HSV hepatitis presenting with distinct risk profiles and clinical manifestations and review the diagnostic and therapeutic challenges associated with this condition. Results: Clinical presentation was nonspecific in all cases, and mucocutaneous lesions were absent or delayed, contributing to diagnostic uncertainty. All patients exhibited marked aminotransferase elevation with relatively modest hyperbilirubinemia (“anicteric hepatitis”), frequently accompanied by cytopenias and coagulopathy. Delayed recognition was associated with rapid clinical deterioration, whereas earlier initiation of intravenous acyclovir was associated with biochemical and clinical improvement. Diagnosis was established using polymerase chain reaction–based detection of HSV DNA in blood and tissue samples. Conclusions: HSV hepatitis remains an under-recognized cause of ALF across a broad spectrum of immunocompromised and seemingly immunocompetent patients. Given the narrow therapeutic window and favourable safety profile of acyclovir, empiric intravenous antiviral therapy should be considered in patients with ALF of indeterminate aetiology while diagnostic testing is pending, particularly in high-risk clinical settings. Increased awareness of this overlooked diagnosis may improve outcomes and prevent avoidable mortality.</p>}},
  author       = {{Mondelli, Mario U. and Ludovisi, Serena and Di Matteo, Angela and Şahin, Gülşen Özkaya and Irving, William}},
  issn         = {{1478-3223}},
  keywords     = {{acute liver failure; acyclovir; hepatitis; herpes simplex virus}},
  language     = {{eng}},
  number       = {{7}},
  publisher    = {{Wiley-Blackwell}},
  series       = {{Liver International}},
  title        = {{Herpes Simplex Virus Hepatitis : An Overlooked Cause of Acute Liver Failure Across a Broad Spectrum of Immunocompromise}},
  url          = {{http://dx.doi.org/10.1111/liv.70756}},
  doi          = {{10.1111/liv.70756}},
  volume       = {{46}},
  year         = {{2026}},
}