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Regulation of KRAS protein expression by miR-544a and KRAS-LCS6 polymorphism in wild-type KRAS sporadic colon adenocarcinoma

Marinović, Sonja ; Škrtić, Anita ; Catela Ivković, Tina LU orcid ; Poljak, Mirko and Kapitanović, Sanja (2021) In Human Cell 34(5). p.1455-1465
Abstract

Colorectal carcinoma (CRC) results from the accumulation of genetic mutations and alterations in signaling pathways. KRAS is mutated in 40% of CRC cases and is involved in increased tumor cells proliferation and survival. Although KRAS mutations are a dominant event in CRC tumorigenesis, increased wild-type KRAS expression has a similar effect on accelerated tumor growth. In this study, we investigated the KRAS status in correlation with clinicopathological features in sporadic CRC and more importantly the role of let-7a-5p and miR-544a-3p in the regulation of wild-type KRAS protein expression in the tumor center (T1) and invasive tumor front (T2). Analysis showed that 39.1% of tumor samples had KRAS mutations. In wild-type KRAS tumors,... (More)

Colorectal carcinoma (CRC) results from the accumulation of genetic mutations and alterations in signaling pathways. KRAS is mutated in 40% of CRC cases and is involved in increased tumor cells proliferation and survival. Although KRAS mutations are a dominant event in CRC tumorigenesis, increased wild-type KRAS expression has a similar effect on accelerated tumor growth. In this study, we investigated the KRAS status in correlation with clinicopathological features in sporadic CRC and more importantly the role of let-7a-5p and miR-544a-3p in the regulation of wild-type KRAS protein expression in the tumor center (T1) and invasive tumor front (T2). Analysis showed that 39.1% of tumor samples had KRAS mutations. In wild-type KRAS tumors, 62.0% were positive for KRAS protein expression and there was a higher percentage of KRAS-positive tumor cells and a higher intensity of immunohistochemical reaction in T2 than in T1 samples. This could not be attributed to differences in KRAS mRNA levels, suggesting regulation via miR-544a-3p expression which was significantly decreased in T2 samples. Furthermore, we demonstrated that tumor samples carrying the KRAS-LCS6 variant allele had significantly higher protein expression of the wild-type KRAS. Our results suggest the role of the KRAS-LCS6 polymorphism and miR-544a-3p expression in the regulation of wild-type KRAS protein expression in sporadic CRC.

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author
; ; ; and
publishing date
type
Contribution to journal
publication status
published
keywords
Colon adenocarcinoma, Immunohistochemistry, KRAS, Let-7a, miR-544a
in
Human Cell
volume
34
issue
5
pages
1455 - 1465
publisher
Springer
external identifiers
  • pmid:34235620
  • scopus:85109418004
ISSN
0914-7470
DOI
10.1007/s13577-021-00576-2
language
English
LU publication?
no
additional info
Publisher Copyright: © 2021, Japan Human Cell Society.
id
aa2bd17d-b04c-4d0f-9947-6ddd3c6f3341
date added to LUP
2026-09-20 00:15:53
date last changed
2026-09-22 03:02:36
@article{aa2bd17d-b04c-4d0f-9947-6ddd3c6f3341,
  abstract     = {{<p>Colorectal carcinoma (CRC) results from the accumulation of genetic mutations and alterations in signaling pathways. KRAS is mutated in 40% of CRC cases and is involved in increased tumor cells proliferation and survival. Although KRAS mutations are a dominant event in CRC tumorigenesis, increased wild-type KRAS expression has a similar effect on accelerated tumor growth. In this study, we investigated the KRAS status in correlation with clinicopathological features in sporadic CRC and more importantly the role of let-7a-5p and miR-544a-3p in the regulation of wild-type KRAS protein expression in the tumor center (T1) and invasive tumor front (T2). Analysis showed that 39.1% of tumor samples had KRAS mutations. In wild-type KRAS tumors, 62.0% were positive for KRAS protein expression and there was a higher percentage of KRAS-positive tumor cells and a higher intensity of immunohistochemical reaction in T2 than in T1 samples. This could not be attributed to differences in KRAS mRNA levels, suggesting regulation via miR-544a-3p expression which was significantly decreased in T2 samples. Furthermore, we demonstrated that tumor samples carrying the KRAS-LCS6 variant allele had significantly higher protein expression of the wild-type KRAS. Our results suggest the role of the KRAS-LCS6 polymorphism and miR-544a-3p expression in the regulation of wild-type KRAS protein expression in sporadic CRC.</p>}},
  author       = {{Marinović, Sonja and Škrtić, Anita and Catela Ivković, Tina and Poljak, Mirko and Kapitanović, Sanja}},
  issn         = {{0914-7470}},
  keywords     = {{Colon adenocarcinoma; Immunohistochemistry; KRAS; Let-7a; miR-544a}},
  language     = {{eng}},
  number       = {{5}},
  pages        = {{1455--1465}},
  publisher    = {{Springer}},
  series       = {{Human Cell}},
  title        = {{Regulation of KRAS protein expression by miR-544a and KRAS-LCS6 polymorphism in wild-type KRAS sporadic colon adenocarcinoma}},
  url          = {{http://dx.doi.org/10.1007/s13577-021-00576-2}},
  doi          = {{10.1007/s13577-021-00576-2}},
  volume       = {{34}},
  year         = {{2021}},
}