Increased levels of CSF total but not oligomeric or phosphorylated forms of alpha-synuclein in patients diagnosed with probable Alzheimer's disease
(2017) In Scientific Reports 7.- Abstract
Several studies reported an association between CSF alpha-synuclein (α-syn) and tau in Alzheimer's disease (AD), and demonstrated the significance of α-syn in improving the diagnostic sensitivity/specificity of classical AD CSF biomarkers. In the current study, we measured CSF levels of different α-syn species in a cohort of AD patients (n = 225) who showed a CSF profile typical of AD at baseline as well as in cognitively intact controls (n = 68). CSF total α-syn (t-α-syn) significantly increased in the AD group (p < 0.0001) compared to controls, while oligomeric- and phosphorylated-Ser129-α-syn did not change significantly. ROC analysis showed a sensitivity of 85% and a specificity of 84% (AUC = 0.88) in distinguishing AD from... (More)
Several studies reported an association between CSF alpha-synuclein (α-syn) and tau in Alzheimer's disease (AD), and demonstrated the significance of α-syn in improving the diagnostic sensitivity/specificity of classical AD CSF biomarkers. In the current study, we measured CSF levels of different α-syn species in a cohort of AD patients (n = 225) who showed a CSF profile typical of AD at baseline as well as in cognitively intact controls (n = 68). CSF total α-syn (t-α-syn) significantly increased in the AD group (p < 0.0001) compared to controls, while oligomeric- and phosphorylated-Ser129-α-syn did not change significantly. ROC analysis showed a sensitivity of 85% and a specificity of 84% (AUC = 0.88) in distinguishing AD from controls. T-α-syn levels correlated positively with tau species in AD group and negatively with baseline MMSE score. Our data support the added value of measurement of CSF α-syn species for further characterization of the CSF AD profile.
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- author
- Majbour, Nour K ; Chiasserini, Davide ; Vaikath, Nishant N. LU ; Eusebi, Paolo ; Tokuda, Takahiko ; van de Berg, Wilma D J ; Parnetti, Lucilla ; Calabresi, Paolo and El-Agnaf, Omar M A
- organization
- publishing date
- 2017-01-10
- type
- Contribution to journal
- publication status
- published
- subject
- in
- Scientific Reports
- volume
- 7
- article number
- 40263
- publisher
- Nature Publishing Group
- external identifiers
-
- pmid:28071698
- wos:000391832100001
- scopus:85009070550
- ISSN
- 2045-2322
- DOI
- 10.1038/srep40263
- language
- English
- LU publication?
- yes
- id
- cb53467a-71f5-4806-bf98-7b56572e057d
- date added to LUP
- 2017-03-02 10:42:35
- date last changed
- 2024-10-28 01:53:43
@article{cb53467a-71f5-4806-bf98-7b56572e057d, abstract = {{<p>Several studies reported an association between CSF alpha-synuclein (α-syn) and tau in Alzheimer's disease (AD), and demonstrated the significance of α-syn in improving the diagnostic sensitivity/specificity of classical AD CSF biomarkers. In the current study, we measured CSF levels of different α-syn species in a cohort of AD patients (n = 225) who showed a CSF profile typical of AD at baseline as well as in cognitively intact controls (n = 68). CSF total α-syn (t-α-syn) significantly increased in the AD group (p < 0.0001) compared to controls, while oligomeric- and phosphorylated-Ser129-α-syn did not change significantly. ROC analysis showed a sensitivity of 85% and a specificity of 84% (AUC = 0.88) in distinguishing AD from controls. T-α-syn levels correlated positively with tau species in AD group and negatively with baseline MMSE score. Our data support the added value of measurement of CSF α-syn species for further characterization of the CSF AD profile.</p>}}, author = {{Majbour, Nour K and Chiasserini, Davide and Vaikath, Nishant N. and Eusebi, Paolo and Tokuda, Takahiko and van de Berg, Wilma D J and Parnetti, Lucilla and Calabresi, Paolo and El-Agnaf, Omar M A}}, issn = {{2045-2322}}, language = {{eng}}, month = {{01}}, publisher = {{Nature Publishing Group}}, series = {{Scientific Reports}}, title = {{Increased levels of CSF total but not oligomeric or phosphorylated forms of alpha-synuclein in patients diagnosed with probable Alzheimer's disease}}, url = {{http://dx.doi.org/10.1038/srep40263}}, doi = {{10.1038/srep40263}}, volume = {{7}}, year = {{2017}}, }