Selective neuroprotective effects of the S18Y polymorphic variant of UCH-L1 in the dopaminergic system
(2012) In Human Molecular Genetics 21(4). p.874-889- Abstract
- Genetic studies have implicated the neuronal ubiquitin C-terminal hydrolase (UCH) protein UCH-L1 in Parkinson's disease (PD) pathogenesis. Moreover, the function of UCH-L1 may be lost in the brains of PD and Alzheimer's disease patients. We have previously reported that the UCH-L1 polymorphic variant S18Y, potentially protective against PD in population studies, demonstrates specific antioxidant functions in cell culture. Albeit genetic, biochemical and neuropathological data support an association between UCH-L1, PD, synaptic degeneration and oxidative stress, the relationship between the dopaminergic system and UCH-L1 status remains obscure. In the current study, we have examined the dopaminergic system of mice lacking endogenous UCH-L1... (More)
- Genetic studies have implicated the neuronal ubiquitin C-terminal hydrolase (UCH) protein UCH-L1 in Parkinson's disease (PD) pathogenesis. Moreover, the function of UCH-L1 may be lost in the brains of PD and Alzheimer's disease patients. We have previously reported that the UCH-L1 polymorphic variant S18Y, potentially protective against PD in population studies, demonstrates specific antioxidant functions in cell culture. Albeit genetic, biochemical and neuropathological data support an association between UCH-L1, PD, synaptic degeneration and oxidative stress, the relationship between the dopaminergic system and UCH-L1 status remains obscure. In the current study, we have examined the dopaminergic system of mice lacking endogenous UCH-L1 protein (gracile axonal dystrophy mice). Our findings show that the lack of wild-type (WT) UCH-L1 does not influence to any significant degree the dopaminergic system at baseline or following injections of the neurotoxin methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP). Furthermore, using a novel intrastriatal adenoviral injection protocol, we have found that mouse nigral neurons retrogradely transduced with S18Y UCH-L1, but not the WT protein, are significantly protected against MPTP toxicity. Overall, these data provide evidence for an antioxidant and neuroprotective effect of the S18Y variant of UCH-L1, but not of the WT protein, in the dopaminergic system, and may have implications for the pathogenesis of PD or related neurodegenerative conditions, in which oxidative stress might play a role. (Less)
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- author
- organization
- publishing date
- 2012
- type
- Contribution to journal
- publication status
- published
- subject
- in
- Human Molecular Genetics
- volume
- 21
- issue
- 4
- pages
- 874 - 889
- publisher
- Oxford University Press
- external identifiers
-
- wos:000299792800013
- scopus:84856347615
- ISSN
- 0964-6906
- DOI
- 10.1093/hmg/ddr521
- language
- English
- LU publication?
- yes
- id
- d56fb286-7c1a-4cc4-a92d-60ae2b2775bf (old id 2416084)
- date added to LUP
- 2016-04-01 11:08:17
- date last changed
- 2022-01-26 05:44:41
@article{d56fb286-7c1a-4cc4-a92d-60ae2b2775bf, abstract = {{Genetic studies have implicated the neuronal ubiquitin C-terminal hydrolase (UCH) protein UCH-L1 in Parkinson's disease (PD) pathogenesis. Moreover, the function of UCH-L1 may be lost in the brains of PD and Alzheimer's disease patients. We have previously reported that the UCH-L1 polymorphic variant S18Y, potentially protective against PD in population studies, demonstrates specific antioxidant functions in cell culture. Albeit genetic, biochemical and neuropathological data support an association between UCH-L1, PD, synaptic degeneration and oxidative stress, the relationship between the dopaminergic system and UCH-L1 status remains obscure. In the current study, we have examined the dopaminergic system of mice lacking endogenous UCH-L1 protein (gracile axonal dystrophy mice). Our findings show that the lack of wild-type (WT) UCH-L1 does not influence to any significant degree the dopaminergic system at baseline or following injections of the neurotoxin methyl-4-phenyl-1,2,3,6-tetrahydropyridine (MPTP). Furthermore, using a novel intrastriatal adenoviral injection protocol, we have found that mouse nigral neurons retrogradely transduced with S18Y UCH-L1, but not the WT protein, are significantly protected against MPTP toxicity. Overall, these data provide evidence for an antioxidant and neuroprotective effect of the S18Y variant of UCH-L1, but not of the WT protein, in the dopaminergic system, and may have implications for the pathogenesis of PD or related neurodegenerative conditions, in which oxidative stress might play a role.}}, author = {{Xilouri, Maria and Kyratzi, Elli and Pitychoutis, Pothitos M. and Papadopoulou-Daifoti, Zoi and Perier, Celine and Vila, Miquel and Maniati, Matina and Ulusoy, Ayse and Kirik, Deniz and Park, David S. and Wada, Keiji and Stefanis, Leonidas}}, issn = {{0964-6906}}, language = {{eng}}, number = {{4}}, pages = {{874--889}}, publisher = {{Oxford University Press}}, series = {{Human Molecular Genetics}}, title = {{Selective neuroprotective effects of the S18Y polymorphic variant of UCH-L1 in the dopaminergic system}}, url = {{http://dx.doi.org/10.1093/hmg/ddr521}}, doi = {{10.1093/hmg/ddr521}}, volume = {{21}}, year = {{2012}}, }