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Small Particles, Big Problems : Polystyrene nanoparticles induce DNA damage, oxidative stress, migration, and mitogenic pathways predominantly in non-malignant lung cells

Ernhofer, Büsra ; Spittler, Andreas ; Ferk, Franziska ; Mišík, Miroslav ; Zylka, Martha Magdalena ; Glatt, Lisa ; Boettiger, Kristiina ; Solta, Anna ; Kirchhofer, Dominik and Timelthaler, Gerald , et al. (2025) In Journal of Hazardous Materials 495.
Abstract

Polystyrene micro- and nanoplastics (PS-MNPs) are emerging environmental pollutants with potential implications for human health. This study investigated the cytotoxic effects of PS particles by using two different sizes of PS-MNPs (0.25 µm and 1 µm) on non-small cell lung cancer (A549, H460), small cell lung cancer (DMS53, H372), and normal lung epithelial (BEAS-2B) cells as well as on human-derived lung organoids. Neither PS-MPs nor PS-NPs interfered with cell viability or proliferation at lower concentrations (< 30 µg/cm2, equivalent to 50 µg/ml). Intracellular kinetic assays revealed that non-malignant (BEAS-2B) lung cells had the strongest turnover of PS-NPs compared to malignant cells. Since PS-NPs exhibited more... (More)

Polystyrene micro- and nanoplastics (PS-MNPs) are emerging environmental pollutants with potential implications for human health. This study investigated the cytotoxic effects of PS particles by using two different sizes of PS-MNPs (0.25 µm and 1 µm) on non-small cell lung cancer (A549, H460), small cell lung cancer (DMS53, H372), and normal lung epithelial (BEAS-2B) cells as well as on human-derived lung organoids. Neither PS-MPs nor PS-NPs interfered with cell viability or proliferation at lower concentrations (< 30 µg/cm2, equivalent to 50 µg/ml). Intracellular kinetic assays revealed that non-malignant (BEAS-2B) lung cells had the strongest turnover of PS-NPs compared to malignant cells. Since PS-NPs exhibited more pronounced cellular effects, additional analyses focused on their impact. Furthermore, we observed significantly increased migration, prolonged S-phase arrest with induced DNA damage, and oxidative stress in non-malignant (BEAS-2B) lung cells. Thus, our data suggest that BEAS-2B cells exhibit the highest sensitivity to PS-NPs. These cells displayed decreased base excision repair capacity and increased activation of survival pathways including AKT and ERK phosphorylation after PS-NP treatment. PS-NP internalization and increase of signal pathways were validated in a physiological lung organoid setting. Our findings suggest that while PS-NPs do not significantly affect the malignant behavior of cancer cells, they could promote malignant features in normal lung cells through the induction of survival pathways, migration, and altered stress response mechanisms.

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organization
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type
Contribution to journal
publication status
published
subject
keywords
DNA damage, lung cancer, lung organoids, polystyrene micro- and nanoplastic, small cell lung cancer
in
Journal of Hazardous Materials
volume
495
article number
139129
publisher
Elsevier
external identifiers
  • pmid:40628207
  • scopus:105009785931
ISSN
0304-3894
DOI
10.1016/j.jhazmat.2025.139129
language
English
LU publication?
yes
id
de0ea3c1-ff86-4422-b08e-da3de5057c6c
date added to LUP
2025-11-04 13:20:08
date last changed
2025-11-18 14:24:29
@article{de0ea3c1-ff86-4422-b08e-da3de5057c6c,
  abstract     = {{<p>Polystyrene micro- and nanoplastics (PS-MNPs) are emerging environmental pollutants with potential implications for human health. This study investigated the cytotoxic effects of PS particles by using two different sizes of PS-MNPs (0.25 µm and 1 µm) on non-small cell lung cancer (A549, H460), small cell lung cancer (DMS53, H372), and normal lung epithelial (BEAS-2B) cells as well as on human-derived lung organoids. Neither PS-MPs nor PS-NPs interfered with cell viability or proliferation at lower concentrations (&lt; 30 µg/cm<sup>2</sup>, equivalent to 50 µg/ml). Intracellular kinetic assays revealed that non-malignant (BEAS-2B) lung cells had the strongest turnover of PS-NPs compared to malignant cells. Since PS-NPs exhibited more pronounced cellular effects, additional analyses focused on their impact. Furthermore, we observed significantly increased migration, prolonged S-phase arrest with induced DNA damage, and oxidative stress in non-malignant (BEAS-2B) lung cells. Thus, our data suggest that BEAS-2B cells exhibit the highest sensitivity to PS-NPs. These cells displayed decreased base excision repair capacity and increased activation of survival pathways including AKT and ERK phosphorylation after PS-NP treatment. PS-NP internalization and increase of signal pathways were validated in a physiological lung organoid setting. Our findings suggest that while PS-NPs do not significantly affect the malignant behavior of cancer cells, they could promote malignant features in normal lung cells through the induction of survival pathways, migration, and altered stress response mechanisms.</p>}},
  author       = {{Ernhofer, Büsra and Spittler, Andreas and Ferk, Franziska and Mišík, Miroslav and Zylka, Martha Magdalena and Glatt, Lisa and Boettiger, Kristiina and Solta, Anna and Kirchhofer, Dominik and Timelthaler, Gerald and Megyesfalvi, Zsolt and Kopatz, Verena and Kovar, Heinrich and Knasmueller, Siegfried and Aigner, Clemens and Kenner, Lukas and Döme, Balazs and Schelch, Karin}},
  issn         = {{0304-3894}},
  keywords     = {{DNA damage; lung cancer; lung organoids; polystyrene micro- and nanoplastic; small cell lung cancer}},
  language     = {{eng}},
  publisher    = {{Elsevier}},
  series       = {{Journal of Hazardous Materials}},
  title        = {{Small Particles, Big Problems : Polystyrene nanoparticles induce DNA damage, oxidative stress, migration, and mitogenic pathways predominantly in non-malignant lung cells}},
  url          = {{http://dx.doi.org/10.1016/j.jhazmat.2025.139129}},
  doi          = {{10.1016/j.jhazmat.2025.139129}},
  volume       = {{495}},
  year         = {{2025}},
}