Therapy for BRAFi-Resistant Melanomas: Is WNT5A the Answer?
(2015) In Cancers 7(3). p.1900-1924- Abstract
- In recent years, scientists have advocated the use of targeted therapies in the form of drugs that modulate genes and proteins that are directly associated with cancer progression and metastasis. Malignant melanoma is a dreadful cancer type that has been associated with the rapid dissemination of primary tumors to multiple sites, including bone, brain, liver and lungs. The discovery that approximately 40%-50% of malignant melanomas contain a mutation in BRAF at codon 600 gave scientists a new approach to tackle this disease. However, clinical studies on patients have shown that although BRAFi (BRAF inhibitors) trigger early anti-tumor responses, the majority of patients later develop resistance to the therapy. Recent studies have shown... (More)
- In recent years, scientists have advocated the use of targeted therapies in the form of drugs that modulate genes and proteins that are directly associated with cancer progression and metastasis. Malignant melanoma is a dreadful cancer type that has been associated with the rapid dissemination of primary tumors to multiple sites, including bone, brain, liver and lungs. The discovery that approximately 40%-50% of malignant melanomas contain a mutation in BRAF at codon 600 gave scientists a new approach to tackle this disease. However, clinical studies on patients have shown that although BRAFi (BRAF inhibitors) trigger early anti-tumor responses, the majority of patients later develop resistance to the therapy. Recent studies have shown that WNT5A plays a key role in enhancing the resistance of melanoma cells to BRAFi. The focus of the current review will be on melanoma development, signaling pathways important to acquired resistance to BRAFi, and why WNT5A inhibitors are attractive candidates to be included in combinatorial therapies for melanoma. (Less)
Please use this url to cite or link to this publication:
https://lup.lub.lu.se/record/8035372
- author
- Prasad, Chandra LU ; Mohapatra, Purusottam LU and Andersson, Tommy LU
- organization
- publishing date
- 2015
- type
- Contribution to journal
- publication status
- published
- subject
- in
- Cancers
- volume
- 7
- issue
- 3
- pages
- 1900 - 1924
- publisher
- MDPI AG
- external identifiers
-
- pmid:26393652
- scopus:84941953633
- pmid:26393652
- wos:000209951200042
- ISSN
- 2072-6694
- DOI
- 10.3390/cancers7030868
- language
- English
- LU publication?
- yes
- id
- e52c4d5e-1b09-4ff1-96cf-942dce80d03a (old id 8035372)
- alternative location
- http://www.ncbi.nlm.nih.gov/pubmed/26393652?dopt=Abstract
- date added to LUP
- 2016-04-04 09:10:29
- date last changed
- 2022-05-09 03:45:33
@article{e52c4d5e-1b09-4ff1-96cf-942dce80d03a, abstract = {{In recent years, scientists have advocated the use of targeted therapies in the form of drugs that modulate genes and proteins that are directly associated with cancer progression and metastasis. Malignant melanoma is a dreadful cancer type that has been associated with the rapid dissemination of primary tumors to multiple sites, including bone, brain, liver and lungs. The discovery that approximately 40%-50% of malignant melanomas contain a mutation in BRAF at codon 600 gave scientists a new approach to tackle this disease. However, clinical studies on patients have shown that although BRAFi (BRAF inhibitors) trigger early anti-tumor responses, the majority of patients later develop resistance to the therapy. Recent studies have shown that WNT5A plays a key role in enhancing the resistance of melanoma cells to BRAFi. The focus of the current review will be on melanoma development, signaling pathways important to acquired resistance to BRAFi, and why WNT5A inhibitors are attractive candidates to be included in combinatorial therapies for melanoma.}}, author = {{Prasad, Chandra and Mohapatra, Purusottam and Andersson, Tommy}}, issn = {{2072-6694}}, language = {{eng}}, number = {{3}}, pages = {{1900--1924}}, publisher = {{MDPI AG}}, series = {{Cancers}}, title = {{Therapy for BRAFi-Resistant Melanomas: Is WNT5A the Answer?}}, url = {{http://dx.doi.org/10.3390/cancers7030868}}, doi = {{10.3390/cancers7030868}}, volume = {{7}}, year = {{2015}}, }