RipD (Rv1566c) from mycobacterium tuberculosis : Adaptation of an NlpC/p60 domain to a non-catalytic peptidoglycan-binding function
(2014) In Biochemical Journal 457(1). p.33-41- Abstract
Enzymes carrying NlpC/p60 domains, for instance RipA and RipB from Mycobacterium tuberculosis, are bacterial peptidoglycan hydrolases that cleave the peptide stems and contribute to cellwall remodelling during cell division.Amember of this protein family, RipD (Rv1566c) from M. tuberculosis described in the present study, displays sequence alterations in the NlpC/p60 catalytic triad and carries a pentapeptide repeat at its C-terminus. Bioinformatics analysis revealed RipD-like proteins in eleven mycobacterial genomes, whereas similar pentapeptide repeats occur in cell-wall-localized bacterial proteins and in a mycobacteriophage. In contrast with previously known members of the NlpC/p60 family, RipD does not show peptidoglycan hydrolase... (More)
Enzymes carrying NlpC/p60 domains, for instance RipA and RipB from Mycobacterium tuberculosis, are bacterial peptidoglycan hydrolases that cleave the peptide stems and contribute to cellwall remodelling during cell division.Amember of this protein family, RipD (Rv1566c) from M. tuberculosis described in the present study, displays sequence alterations in the NlpC/p60 catalytic triad and carries a pentapeptide repeat at its C-terminus. Bioinformatics analysis revealed RipD-like proteins in eleven mycobacterial genomes, whereas similar pentapeptide repeats occur in cell-wall-localized bacterial proteins and in a mycobacteriophage. In contrast with previously known members of the NlpC/p60 family, RipD does not show peptidoglycan hydrolase activity, which is consistent with the sequence alterations at the catalytic site. A strong interaction of the catalytically inactive core domain with peptidoglycan is however retained, presenting the first example of the NlpC/p60 domains that evolved to a non-catalytic peptidoglycan-binding function. Full-length RipD carrying the C-terminal repeat shows, however, a decrease in binding affinity to peptidoglycan, suggesting that the C-terminal tail modulates the interaction with bacterial cell wall components. The pentapeptide repeat at the C-terminus does not adopt a defined secondary structure in solution which is in accordance with results from the 1.17 Å (1 Å=0.1 nm) crystal structure of the protein carrying two repeat units.
(Less)
- author
- Böth, Dominic
; Steiner, Eva Maria
LU
; Izumi, Atsushi ; Schneider, Gunter and Schnell, Robert
- publishing date
- 2014-01-01
- type
- Contribution to journal
- publication status
- published
- subject
- keywords
- Catalytic site, Cell wall, Mycobacterium tuberculosis, NlpC/p60, Peptidoglycan binding, Proline-rich, Protein repeat, Protein structure
- in
- Biochemical Journal
- volume
- 457
- issue
- 1
- pages
- 33 - 41
- publisher
- Portland Press
- external identifiers
-
- pmid:24107184
- scopus:84890039458
- ISSN
- 0264-6021
- DOI
- 10.1042/BJ20131227
- language
- English
- LU publication?
- no
- id
- f19b27c8-e815-449d-bc18-688352fb5c10
- date added to LUP
- 2024-12-11 10:45:14
- date last changed
- 2025-04-04 15:21:48
@article{f19b27c8-e815-449d-bc18-688352fb5c10, abstract = {{<p>Enzymes carrying NlpC/p60 domains, for instance RipA and RipB from Mycobacterium tuberculosis, are bacterial peptidoglycan hydrolases that cleave the peptide stems and contribute to cellwall remodelling during cell division.Amember of this protein family, RipD (Rv1566c) from M. tuberculosis described in the present study, displays sequence alterations in the NlpC/p60 catalytic triad and carries a pentapeptide repeat at its C-terminus. Bioinformatics analysis revealed RipD-like proteins in eleven mycobacterial genomes, whereas similar pentapeptide repeats occur in cell-wall-localized bacterial proteins and in a mycobacteriophage. In contrast with previously known members of the NlpC/p60 family, RipD does not show peptidoglycan hydrolase activity, which is consistent with the sequence alterations at the catalytic site. A strong interaction of the catalytically inactive core domain with peptidoglycan is however retained, presenting the first example of the NlpC/p60 domains that evolved to a non-catalytic peptidoglycan-binding function. Full-length RipD carrying the C-terminal repeat shows, however, a decrease in binding affinity to peptidoglycan, suggesting that the C-terminal tail modulates the interaction with bacterial cell wall components. The pentapeptide repeat at the C-terminus does not adopt a defined secondary structure in solution which is in accordance with results from the 1.17 Å (1 Å=0.1 nm) crystal structure of the protein carrying two repeat units.</p>}}, author = {{Böth, Dominic and Steiner, Eva Maria and Izumi, Atsushi and Schneider, Gunter and Schnell, Robert}}, issn = {{0264-6021}}, keywords = {{Catalytic site; Cell wall; Mycobacterium tuberculosis; NlpC/p60; Peptidoglycan binding; Proline-rich; Protein repeat; Protein structure}}, language = {{eng}}, month = {{01}}, number = {{1}}, pages = {{33--41}}, publisher = {{Portland Press}}, series = {{Biochemical Journal}}, title = {{RipD (Rv1566c) from mycobacterium tuberculosis : Adaptation of an NlpC/p60 domain to a non-catalytic peptidoglycan-binding function}}, url = {{http://dx.doi.org/10.1042/BJ20131227}}, doi = {{10.1042/BJ20131227}}, volume = {{457}}, year = {{2014}}, }