1 – 14 of 14
- show: 50
- |
- sort: year (new to old)
Close
Embed this list
<iframe src=" "
width=" "
height=" "
allowtransparency="true"
frameborder="0">
</iframe>
- 2019
-
Mark
Integrin α10, a novel therapeutic target in glioblastoma, regulates cell migration, proliferation, and survival
(
- Contribution to journal › Article
- 2018
-
Mark
BAP1 induces cell death via interaction with 14-3-3 in neuroblastoma article
(
- Contribution to journal › Article
- 2017
-
Mark
NLK-mediated phosphorylation of HDAC1 negatively regulates Wnt signaling
(
- Contribution to journal › Article
- 2016
-
Mark
Putative role of SUMOylation in controlling the activity of deubiquitinating enzymes in cancer.
(
- Contribution to journal › Scientific review
-
Mark
Nemo-like kinase regulates the expression of vascular endothelial growth factor (VEGF) in alveolar epithelial cells
(
- Contribution to journal › Article
-
Mark
CYLD and SUMO in neuroblastoma therapy
(
- Contribution to journal › Article
-
Mark
Molecular characterization of protein kinase C delta (PKCδ)-Smac interactions
(
- Contribution to journal › Article
-
Mark
PKC activation sensitizes basal-like breast cancer cell lines to Smac mimetics
(
- Contribution to journal › Article
- 2015
-
Mark
Early diagnostic value of Bcl-3 localization in colorectal cancer.
(
- Contribution to journal › Article
-
Mark
Deubiquitinating activity of CYLD is impaired by SUMOylation in neuroblastoma cells.
(
- Contribution to journal › Article
- 2014
-
Mark
Association of nuclear-localized nemo-like kinase with heat-shock protein 27 inhibits apoptosis in human breast cancer cells.
(
- Contribution to journal › Article
- 2012
-
Mark
Identification of a novel protein kinase Cδ-Smac complex that dissociates during paclitaxel-induced cell death.
(
- Contribution to journal › Article
- 2011
-
Mark
Tumor Suppressor Function of CYLD in Nonmelanoma Skin Cancer.
(
- Contribution to journal › Article
- 2009
-
Mark
Protein Kinase C delta Supports Survival of MDA-MB-231 Breast Cancer Cells by Suppressing the ERK1/2 Pathway
(
- Contribution to journal › Article