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- 2013
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Mark
Loss of chromosomes is the primary event in near-haploid and low hypodiploid acute lymphoblastic leukemia.
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- Contribution to journal › Article
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Mark
High modal number and triple trisomies are highly correlated favorable factors in childhood B-cell precursor high hyperdiploid acute lymphoblastic leukemia treated according to the NOPHO ALL 1992/2000 protocols
(
- Contribution to journal › Article
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Mark
Distinct patterns of gained chromosomes in high hyperdiploid acute lymphoblastic leukemia with t(1;19)(q23;p13), t(9;22)(q34;q22) or MLL rearrangements.
(
- Contribution to journal › Letter
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Mark
Partial 17q gain resulting from isochromosomes, unbalanced translocations, and complex rearrangements is associated with gene overexpression, older age, and shorter overall survival in high hyperdiploid childhood acute lymphoblastic leukemia.
(
- Contribution to journal › Letter
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Mark
Genomic Heterogeneity in Acute Leukemia.
(
- Contribution to journal › Article
- 2012
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Mark
Retained heterodisomy is associated with high gene expression in hyperhaploid inflammatory leiomyosarcoma.
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- Contribution to journal › Article
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Mark
High frequency of BTG1 deletions in acute lymphoblastic leukemia in children with down syndrome.
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- Contribution to journal › Article
- 2011
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Mark
Clonal Evolution through Loss of Chromosomes and Subsequent Polyploidization in Chondrosarcoma.
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- Contribution to journal › Article
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Mark
The insulin receptor substrate 4 gene (IRS4) is mutated in paediatric T-cell acute lymphoblastic leukaemia.
(
- Contribution to journal › Letter
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Mark
t(9;11)(p22;p15) [NUP98/PSIP1] is a poor prognostic marker associated with de novo acute myeloid leukaemia expressing both mature and immature surface antigens.
(
- Contribution to journal › Letter