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- 2005
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Mark
Structural and numerical chromosome changes in colon cancer develop through telomere-mediated anaphase bridges, not through mitotic multipolarity.
(
- Contribution to journal › Article
- 2004
-
Mark
Telomere shortening and mitotic dysfunction generate cytogenetic heterogeneity in a subgroup of renal cell carcinomas.
(
- Contribution to journal › Article
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Mark
Genome-wide array-based comparative genomic hybridization reveals multiple amplification targets and novel homozygous deletions in pancreatic carcinoma cell lines.
(
- Contribution to journal › Article
- 2003
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Mark
Pancreatic carcinoma cell lines with SMAD4 inactivation show distinct expression responses to TGFB1.
(
- Contribution to journal › Article
- 2002
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Mark
Frequent amplification of 8q24, 11q, 17q, and 20q-specific genes in pancreatic cancer
(
- Contribution to journal › Article
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Mark
Detailed genomic mapping and expression analyses of 12p amplifications in pancreatic carcinomas reveal a 3.5-Mb target region for amplification.
(
- Contribution to journal › Article
- 2001
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Mark
Extensive cytogenetic heterogeneity in a benign retroperitoneal schwannoma
(
- Contribution to journal › Article
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Mark
Altered expression of TGFB receptors and mitogenic effects of TGFB in pancreatic carcinomas
(
- Contribution to journal › Article
- 2000
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Mark
Characterization of genomically amplified segments using PCR: optimizing relative-PCR for reliable and simple gene expression and gene copy analyses
(
- Contribution to journal › Article
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Mark
Locus-specific multifluor FISH analysis allows physical characterization of complex chromosome abnormalities in neoplasia
(
- Contribution to journal › Article