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- 2008
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Mark
Treatment of migraine attacks based on the interaction with the trigemino-cerebrovascular system.
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- Contribution to journal › Article
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Mark
PKC and MAPK signalling pathways regulate vascular endothelin receptor expression
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- Contribution to journal › Article
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Mark
Lipid soluble smoke particles up-regulate vascular smooth muscle ETB receptors via activation of mitogen activating protein kinases and NF-kappaB pathways.
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- Contribution to journal › Article
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Mark
Subtype activation and interaction of protein kinase C and mitogen-activated protein kinase controlling receptor expression in cerebral arteries and microvessels after subarachnoid hemorrhage
(
- Contribution to journal › Article
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Mark
Subarachnoid hemorrhage induces upregulation of vascular receptors and reduction in rCBF via an ERKI/2 mechanism
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- Chapter in Book/Report/Conference proceeding › Paper in conference proceeding
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Mark
Transcriptional down-regulation of thromboxane A2 receptor expression via activation of intracellular MAPK ERK1/2 and p38/NF-kappa B inflammatory signal
2008) 18th Scientific Meeting of the European-Society-of-Hypertension/22nd Scientific Meeting of the International-Society-of-Hypertension In Journal of Hypertension 26(Suppl. 1). p.24-25(
- Contribution to journal › Published meeting abstract
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Mark
Changes in perfusion pressure alter vascular GPCR expression in rat mesenteric artery
2008) 18th Scientific Meeting of the European-Society-of-Hypertension/22nd Scientific Meeting of the International-Society-of-Hypertension In Journal of Hypertension 26(Suppl. 1). p.309-309(
- Contribution to journal › Published meeting abstract
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Mark
Enhanced external counter pulsation in treatment of refractory angina pectoris: two year outcome and baseline factors associated with treatment failure
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- Contribution to journal › Article
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Mark
Vascular endothelin type B receptor upregulation correlates with activation of inflammatory transcriptional factor NF-kappaB
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- Contribution to journal › Published meeting abstract
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Mark
Elevated intracranial pressure or subarachnoid blood responsible for reduction in cerebral blood flow after SAH
(
- Chapter in Book/Report/Conference proceeding › Paper in conference proceeding