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- 2006
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Mark
The contact system-a novel branch of innate immunity generating antibacterial peptides.
(
- Contribution to journal › Article
- 2004
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Mark
Heparin binding protein is increased in chronic leg ulcer fluid and released from granulocytes by secreted products of Pseudomonas aeruginosa.
(
- Contribution to journal › Article
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Mark
M Protein, a Classical Bacterial Virulence Determinant, Forms Complexes with Fibrinogen that Induce Vascular Leakage
(
- Contribution to journal › Article
- 2003
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Mark
The conversion of fibrinogen to fibrin at the surface of curliated E. coli bacteria leads to the generation of proinflammatory fibrinopeptides.
(
- Contribution to journal › Article
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Mark
Superantigens from Staphylococcus aureus induce procoagulant activity and monocyte tissue factor expression in whole blood and mononuclear cells via IL-1beta.
(
- Contribution to journal › Article
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Mark
Contact activation by pathogenic bacteria: a virulence mechanism contributing to the pathophysiology of sepsis.
(
- Contribution to journal › Article
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Mark
Interactions between surface proteins of Streptococcus pyogenes and coagulation factors modulate clotting of human plasma.
(
- Contribution to journal › Article
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Mark
Superantigens from Staphylococcus aureus induce procoagulant activity and monocyte tissue factor expression in whole blood and mononuclear cells via IL-1 beta
(
- Contribution to journal › Article
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Mark
Modulation of membrane traffic by microorganisms.
(
- Contribution to journal › Article
- 2002
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Mark
Identification of two protein-binding and functional regions of curli, a surface organelle and virulence determinant of Escherichia coli
(
- Contribution to journal › Article