1 – 10 of 12
- show: 10
- |
- sort: year (new to old)
Close
Embed this list
<iframe src=" "
width=" "
height=" "
allowtransparency="true"
frameborder="0">
</iframe>
- 2016
-
Mark
CART is overexpressed in human type 2 diabetic islets and inhibits glucagon secretion and increases insulin secretion
(
- Contribution to journal › Article
-
Mark
Serotonin (5-HT) receptor 2b activation augments glucose-stimulated insulin secretion in human and mouse islets of Langerhans.
(
- Contribution to journal › Article
- 2015
-
Mark
Altered serotonin (5-HT) 1D and 2A receptor expression may contribute to defective insulin and glucagon secretion in human type 2 diabetes.
(
- Contribution to journal › Article
- 2012
-
Mark
Impact of an Exercise Intervention on DNA Methylation in Skeletal Muscle From First-Degree Relatives of Patients With Type 2 Diabetes.
2012) In Diabetes(
- Contribution to journal › Article
- 2011
-
Mark
Nesfatin-1 stimulates glucagon and insulin secretion and beta cell NUCB2 is reduced in human type 2 diabetic subjects.
(
- Contribution to journal › Article
- 2010
-
Mark
Nesfatin-1 stimulates insulin secretion, inhibits glucagon secretion and is expressed in human and rodent beta cells
2010) 46th Annual Meeting of the European-Association-for-the- Study-of-Diabetes (EASD) In Diabetologia 53(Suppl. 1). p.548-548(
- Contribution to journal › Published meeting abstract
-
Mark
Stimulation of 5-HT1a receptor decreased insulin secretion in islets of Langerhans from mice and humans
2010) 46th Annual Meeting of the European-Association-for-the- Study-of-Diabetes (EASD) In Diabetologia 53(Suppl. 1). p.547-547(
- Contribution to journal › Published meeting abstract
-
Mark
Apelin is expressed in human and rodent islets and regulates insulin secretion
(
- Contribution to journal › Published meeting abstract
-
Mark
Apelin is a novel islet peptide.
(
- Contribution to journal › Article
- 2009
-
Mark
A beta cell-specific knockout of hormone-sensitive lipase in mice results in hyperglycaemia and disruption of exocytosis.
(
- Contribution to journal › Article