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- 2007
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Mark
An extensive polymerase chain reaction-allele-specific polymorphism strategy for clinical ABO blood group genotyping that avoids potential errors caused by null, subgroup, and hybrid alleles
(
- Contribution to journal › Article
-
Mark
Structural basis for red cell phenotypic changes in newly identified, naturally occurring subgroup mutants of the human blood group B glycosyltransferase.
(
- Contribution to journal › Article
- 2005
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Mark
The A phenotype in the ABO blood group system is due to a splice-site mutation in a hybrid between a new O-like allelic lineage and the A allele.
(
- Contribution to journal › Article
-
Mark
New and unusual O alleles at the ABO locus are implicated in unexpected blood group phenotypes.
(
- Contribution to journal › Article