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- 2011
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Mark
Inhibition of cerebrovascular raf activation attenuates cerebral blood flow and prevents upregulation of contractile receptors after subarachnoid hemorrhage
(
- Contribution to journal › Article
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Mark
The role of tumor necrosis factor-alpha and TNF-alpha receptors in cerebral arteries following cerebral ischemia in rat
(
- Contribution to journal › Article
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Mark
Blockade of the MEK/ERK pathway with a raf inhibitor prevents activation of pro-inflammatory mediators in cerebral arteries and reduction in cerebral blood flow after subarachnoid hemorrhage in a rat model.
2011) In Journal of cerebral blood flow and metabolism : official journal of the International Society of Cerebral Blood Flow and Metabolism May 4. p.144-154(
- Contribution to journal › Article
- 2010
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Mark
Subarachnoid Hemorrhage Induces Enhanced Expression of Thromboxane A(2) Receptors in Rat Cerebral Arteries.
(
- Contribution to journal › Article
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Mark
Cerebral ischemia induces microvascular pro-inflammatory cytokine expression via the MEK/ERK pathway.
(
- Contribution to journal › Article
- 2009
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Mark
RAF inhibition prevents activation of pro-inflammatory mediators in cerebral arteries after subarachnoid hemorrhage
2009) 24th International Symposium on Cerebral Blood Flow and Metabolism/9th International Conference on Quantification of Brain Function with PET In Journal of Cerebral Blood Flow and Metabolism 29. p.305-305(
- Contribution to journal › Published meeting abstract
-
Mark
Enhanced cerebrovascular expression of matrix metalloproteinase-9 and tissue inhibitor of metalloproteinase-1 via the MEK/ERK pathway during cerebral ischemia in the rat.
(
- Contribution to journal › Article
- 2008
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Mark
Enhanced expressions of microvascular smooth muscle receptors after focal cerebral ischemia occur via the MAPK MEK/ERK pathway.
(
- Contribution to journal › Article
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Mark
Enhanced expression of contractile endothelin ET(B) receptors in rat coronary artery after organ culture.
(
- Contribution to journal › Article
- 2007
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Mark
Cooperative effect of angiotensin AT, and endothelin ETA receptor antagonism limits the brain damage after ischemic stroke in rat
(
- Contribution to journal › Article
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