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- 2004
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Mark
M Protein, a Classical Bacterial Virulence Determinant, Forms Complexes with Fibrinogen that Induce Vascular Leakage
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- Contribution to journal › Article
- 2003
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Mark
Interactions between surface proteins of Streptococcus pyogenes and coagulation factors modulate clotting of human plasma.
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- Contribution to journal › Article
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Mark
Modulation of membrane traffic by microorganisms.
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- Contribution to journal › Article
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Mark
Contact activation by pathogenic bacteria: a virulence mechanism contributing to the pathophysiology of sepsis.
(
- Contribution to journal › Article
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Mark
Superantigens from Staphylococcus aureus induce procoagulant activity and monocyte tissue factor expression in whole blood and mononuclear cells via IL-1 beta
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- Contribution to journal › Article
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Mark
The conversion of fibrinogen to fibrin at the surface of curliated E. coli bacteria leads to the generation of proinflammatory fibrinopeptides.
(
- Contribution to journal › Article
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Mark
Superantigens from Staphylococcus aureus induce procoagulant activity and monocyte tissue factor expression in whole blood and mononuclear cells via IL-1beta.
(
- Contribution to journal › Article
- 2002
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Mark
Contact-system activation in children with vasculitis.
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- Contribution to journal › Article
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Mark
Secretion of heparin-binding protein from human neutrophils is determined by its localization in azurophilic granules and secretory vesicles.
(
- Contribution to journal › Article
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Mark
Peptidoglycan from Staphylococcus aureus induces tissue factor expression and procoagulant activity in human monocytes.
(
- Contribution to journal › Article