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- 2010
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Mark
Polymorphisms in CACNA1D affect insulin release and channel expression and associate with type 2 diabetes
2010) 46th Annual Meeting of the European-Association-for-the- Study-of-Diabetes (EASD) In Diabetologia 53(Suppl. 1). p.296-296(
- Contribution to journal › Published meeting abstract
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Mark
GLP-1 inhibits and adrenaline stimulates glucagon release by differential modulation of N- and L-type Ca2+ channel-dependent exocytosis.
(
- Contribution to journal › Article
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Mark
Molecular Function of TCF7L2: Consequences of TCF7L2 Splicing for Molecular Function and Risk for Type 2 Diabetes.
(
- Contribution to journal › Article
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Mark
Overexpression of Alpha2A-Adrenergic Receptors Contributes to Type 2 Diabetes.
(
- Contribution to journal › Article
- 2009
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Mark
Hyperactivity in adrenergic signalling via alpha2A receptors contributes to human type 2 diabetes
2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.108-108(
- Contribution to journal › Published meeting abstract
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Mark
The impact of differential splicing of TCF7L2 on target gene expression in human islet of Langerhans
2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.78-78(
- Contribution to journal › Published meeting abstract
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Mark
Evaluation of chloride channel-3 as a potential target for TCF7L2-dependent impairment of insulin secretion
2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.332-332(
- Contribution to journal › Published meeting abstract
-
Mark
Complement factors are expressed in pancreatic islets and affect glucose induced insulin secretion
2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.455-455(
- Contribution to journal › Published meeting abstract
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Mark
Glutaredoxin-1 mediates NADPH-dependent stimulation of calcium-dependent insulin secretion.
(
- Contribution to journal › Article
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Mark
Autologous hematopoietic stem cell transplantation in type 1-diabetes.
(
- Contribution to journal › Article