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- 2010
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Mark
Pseudomonas aeruginosa CRASP-2 is a surface protein that uses the human terminal complement regulator vitronectin for protection against complement-mediated attack
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- Contribution to journal › Published meeting abstract
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Mark
Analysis of binding sites on complement Factor I that are required for its activity
(
- Contribution to journal › Published meeting abstract
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Mark
Ligands for C1q and factor H on the surface of apoptotic cells
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- Contribution to journal › Published meeting abstract
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Mark
Proteases of periodontal pathogens Porphyromonas gingivalis, Tannerella forsythia and Prevotella intermedia target all pathways of the complement system
(
- Contribution to journal › Published meeting abstract
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Mark
The NC4 domain of the cartilage-specific collagen IX is a novel complement inhibitor
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- Contribution to journal › Published meeting abstract
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Mark
CRASP2 of Candida albicans binds C4b-binding protein and mediates fungal contact with human endothelial cells
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- Contribution to journal › Published meeting abstract
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Mark
Inflammation and joint disease: Molecules/fragments that are released upon cartilage breakdown can activate complement and provide a new diagnostics for pathological complement activation
(
- Contribution to journal › Published meeting abstract
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Mark
Analysis of genes coding for CD46, CD55 and C4b-binding protein in patients with idiopathic, recurrent, spontaneous pregnancy loss
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- Contribution to journal › Published meeting abstract
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Mark
Sensitive and specific assays for C3 nephritic factors permit dissection of mechanisms underlying complement dysregulation
(
- Contribution to journal › Published meeting abstract
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Mark
Inter alpha inhibitor (IaI) as novel inhibitor of human complement system
(
- Contribution to journal › Published meeting abstract