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- 2024
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Mark
Interplay between glypican-1, amyloid-β and tau phosphorylation in human neural stem cells
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- Contribution to journal › Article
- 2023
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Mark
Interplay between APP and glypican-1 processing and α-synuclein aggregation in undifferentiated and differentiated human neural progenitor cells
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- Contribution to journal › Article
- 2022
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Mark
Complex modulation of cytokine-induced α-synuclein aggregation by glypican-1-derived heparan sulfate in neural cells
(
- Contribution to journal › Article
- 2021
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Mark
Reversal of apolipoprotein E4-dependent or chemical-induced accumulation of APP degradation products by vitamin C-induced release of heparan sulfate from glypican-1
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- Contribution to journal › Article
- 2020
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Mark
Proinflammatory cytokines induce accumulation of glypican-1-derived heparan sulfate and the C-terminal fragment of β-cleaved APP in autophagosomes of dividing neuronal cells
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- Contribution to journal › Article
- 2019
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Mark
The cyanobacterial neurotoxin β-N-methylamino-L-alanine prevents addition of heparan sulfate to glypican-1 and increases processing of amyloid precursor protein in dividing neuronal cells
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- Contribution to journal › Article
- 2018
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Mark
Common traffic routes for imported spermine and endosomal glypican-1-derived heparan sulfate in fibroblasts
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- Contribution to journal › Article
- 2017
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Mark
Cytochrome b561, copper, β-cleaved amyloid precursor protein and niemann-pick C1 protein are involved in ascorbate-induced release and membrane penetration of heparan sulfate from endosomal S-nitrosylated glypican-1
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- Contribution to journal › Article
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Mark
Nucleolin is a nuclear target of heparan sulfate derived from glypican-1
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- Contribution to journal › Article
- 2016
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Mark
Hypoxia induces NO-dependent release of heparan sulfate in fibroblasts from the Alzheimer mouse Tg2576 by activation of nitrite reduction.
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- Contribution to journal › Article
- 2015
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Mark
Rapid nuclear transit and impaired degradation of amyloid beta and glypican-1-derived heparan sulfate in Tg2576 mouse fibroblasts.
(
- Contribution to journal › Article
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Mark
Suppression of glypican-1 autodegradation by NO-deprivation correlates with nuclear accumulation of amyloid beta in normal fibroblasts.
(
- Contribution to journal › Article
- 2014
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Mark
APP/APLP2 expression is required to initiate endosome-nucleus-autophagosome trafficking of glypican-1-derived heparan sulfate.
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- Contribution to journal › Article
- 2013
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Mark
Non-toxic amyloid beta formed in the presence of glypican-1 or its deaminatively generated heparan sulfate degradation products
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- Contribution to journal › Article
- 2012
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Mark
Non-conserved, S-nitrosylated cysteines in glypican-1 react with N-unsubstituted glucosamines in heparan sulfate and catalyze deaminative cleavage.
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- Contribution to journal › Article
- 2011
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Mark
Suppression of amyloid beta a11-immunoreactivity by vitamin C: possible role of heparan sulfate oligosaccharides derived from glypican-1 by ascorbate-induced, no-catalyzed degradation.
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- Contribution to journal › Article
- 2010
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Mark
Attenuation of tumor growth by formation of antiproliferative glycosaminoglycans correlates with low acetylation of histone H3.
(
- Contribution to journal › Article
- 2009
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Mark
Potentiation of naphthoxyloside cytotoxicity on human tumor cells by difluoromethylornithine and spermine-NONOate.
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- Contribution to journal › Article
- 2008
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Mark
Involvement of glypican-1 autoprocessing in scrapie infection
(
- Contribution to journal › Article
- 2007
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Mark
Heparan sulfate degradation products can associate with oxidized proteins and proteasomes.
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- Contribution to journal › Article