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- 2024
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Mark
Airway hyperresponsiveness correlates with airway TSLP in asthma independent of eosinophilic inflammation
2024) In Journal of Allergy and Clinical Immunology(
- Contribution to journal › Article
- 2023
-
Mark
Airway hyperresponsiveness reflects corticosteroid-sensitive mast cell involvement across asthma phenotypes
(
- Contribution to journal › Article
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Mark
Clonal evolution and stereotyped sequences of human IgE lineages in aeroallergen-specific immunotherapy
(
- Contribution to journal › Article
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Mark
Plasma proteins defending infected, intact nasal mucosa
(
- Contribution to journal › Letter
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Mark
Eosinophils and tissue remodeling : Relevance to airway disease
(
- Contribution to journal › Scientific review
- 2022
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Mark
Epithelial repair in asthma : Defect or exaggerated?
(
- Contribution to journal › Letter
- 2021
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Mark
Allergome-wide peptide microarrays enable epitope deconvolution in allergen-specific immunotherapy
(
- Contribution to journal › Article
- 2020
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Mark
Gain-of-function mutation in complement C2 protein identified in a patient with aHUS
(
- Contribution to journal › Letter
- 2019
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Mark
Airways exudation of plasma macromolecules : Innate defense, epithelial regeneration, and asthma
(
- Contribution to journal › Article
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Mark
IL-33 drives influenza-induced asthma exacerbations by halting innate and adaptive antiviral immunity
(
- Contribution to journal › Article
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Mark
The SQ tree SLIT-tablet is highly effective and well tolerated : Results from a randomized, double-blind, placebo-controlled phase III trial
(
- Contribution to journal › Article
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Mark
IL-33 blockade affects mediators of persistence and exacerbation in a model of chronic airway inflammation
(
- Contribution to journal › Article
- 2018
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Mark
Toll-like receptor 3 blockade in rhinovirus-induced experimental asthma exacerbations : A randomized controlled study
(
- Contribution to journal › Article
-
Mark
Lytic eosinophils produce extracellular DNA traps as well as free eosinophil granules
(
- Contribution to journal › Debate/Note/Editorial
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Mark
Substance P represents a novel first-line defense mechanism in the nose
(
- Contribution to journal › Article