Genetic characterization of amyloid-β and tau network spread
(2018) In Nature Medicine 24. p.1790-1792- Abstract
Protein-specific and shared genetic pathways influence the spread of amyloid-β and tau pathology. Hence, distinct gene expression profiles may induce regional vulnerability of the human cortex to the specific proteinopathies of Alzheimer’s disease.
Please use this url to cite or link to this publication:
https://lup.lub.lu.se/record/6ffc540e-82d7-4e88-88c2-7a226e541b29
- author
- Ossenkoppele, Rik LU and Hansson, Oskar LU
- organization
- publishing date
- 2018
- type
- Contribution to journal
- publication status
- published
- subject
- in
- Nature Medicine
- volume
- 24
- pages
- 1790 - 1792
- publisher
- Nature Publishing Group
- external identifiers
-
- scopus:85058052169
- pmid:30510252
- ISSN
- 1078-8956
- DOI
- 10.1038/s41591-018-0277-2
- language
- English
- LU publication?
- yes
- id
- 6ffc540e-82d7-4e88-88c2-7a226e541b29
- date added to LUP
- 2019-01-02 08:49:57
- date last changed
- 2024-06-11 01:50:36
@article{6ffc540e-82d7-4e88-88c2-7a226e541b29, abstract = {{<p>Protein-specific and shared genetic pathways influence the spread of amyloid-β and tau pathology. Hence, distinct gene expression profiles may induce regional vulnerability of the human cortex to the specific proteinopathies of Alzheimer’s disease.</p>}}, author = {{Ossenkoppele, Rik and Hansson, Oskar}}, issn = {{1078-8956}}, language = {{eng}}, pages = {{1790--1792}}, publisher = {{Nature Publishing Group}}, series = {{Nature Medicine}}, title = {{Genetic characterization of amyloid-β and tau network spread}}, url = {{http://dx.doi.org/10.1038/s41591-018-0277-2}}, doi = {{10.1038/s41591-018-0277-2}}, volume = {{24}}, year = {{2018}}, }