New and Effective Inhibitor of Class I HDACs, Eimbinostat, Reduces the Growth of Hematologic Cancer Cells and Triggers Apoptosis
(2025) In Pharmaceutics 17(4).- Abstract
Background: Histone deacetylases (HDACs) are critical epigenetic modulators involved in regulating various molecular mechanisms essential for cell development and growth. Alterations in HDAC activity have been linked to the progression of numerous cancers, including lymphoma. Over the past decade, the FDA has approved several HDAC inhibitors for lymphoma treatment, leading to heightened interest in this emerging class of drugs. Methods: In our research, we developed a novel HDAC inhibitor that exhibits high selectivity for class I HDACs. Results: Our in vitro findings indicate that treating lymphoma/leukemia cells with this inhibitor results in a marked suppression of cell growth and promotes apoptosis, while leaving the cell cycle... (More)
Background: Histone deacetylases (HDACs) are critical epigenetic modulators involved in regulating various molecular mechanisms essential for cell development and growth. Alterations in HDAC activity have been linked to the progression of numerous cancers, including lymphoma. Over the past decade, the FDA has approved several HDAC inhibitors for lymphoma treatment, leading to heightened interest in this emerging class of drugs. Methods: In our research, we developed a novel HDAC inhibitor that exhibits high selectivity for class I HDACs. Results: Our in vitro findings indicate that treating lymphoma/leukemia cells with this inhibitor results in a marked suppression of cell growth and promotes apoptosis, while leaving the cell cycle unaffected. Conclusions: We propose that our new inhibitor, named eimbinostat, holds significant promise as a potential therapeutic agent for the treatment of hematologic malignancies such as lymphoma or leukemia.
(Less)
- author
- organization
- publishing date
- 2025-04
- type
- Contribution to journal
- publication status
- published
- subject
- keywords
- apoptosis, belinostat, cell cycle, epigenetic, HDACs, leukemia, lymphoma
- in
- Pharmaceutics
- volume
- 17
- issue
- 4
- article number
- 416
- publisher
- MDPI AG
- external identifiers
-
- scopus:105003552914
- pmid:40284412
- ISSN
- 1999-4923
- DOI
- 10.3390/pharmaceutics17040416
- language
- English
- LU publication?
- yes
- additional info
- Publisher Copyright: © 2025 by the authors.
- id
- 57d04857-26be-4d42-8729-361d08f88439
- date added to LUP
- 2025-08-12 10:13:22
- date last changed
- 2025-08-13 03:00:09
@article{57d04857-26be-4d42-8729-361d08f88439, abstract = {{<p>Background: Histone deacetylases (HDACs) are critical epigenetic modulators involved in regulating various molecular mechanisms essential for cell development and growth. Alterations in HDAC activity have been linked to the progression of numerous cancers, including lymphoma. Over the past decade, the FDA has approved several HDAC inhibitors for lymphoma treatment, leading to heightened interest in this emerging class of drugs. Methods: In our research, we developed a novel HDAC inhibitor that exhibits high selectivity for class I HDACs. Results: Our in vitro findings indicate that treating lymphoma/leukemia cells with this inhibitor results in a marked suppression of cell growth and promotes apoptosis, while leaving the cell cycle unaffected. Conclusions: We propose that our new inhibitor, named eimbinostat, holds significant promise as a potential therapeutic agent for the treatment of hematologic malignancies such as lymphoma or leukemia.</p>}}, author = {{Spirin, Pavel and Vedernikova, Valeria and Volkava, Tatsiana and Morozov, Alexey and Kleymenova, Alla and Zemskaya, Anastasia and Shyrokova, Lena and Porozov, Yuri and Glumakova, Ksenia and Lebedev, Timofey and Kozlov, Maxim and Prassolov, Vladimir}}, issn = {{1999-4923}}, keywords = {{apoptosis; belinostat; cell cycle; epigenetic; HDACs; leukemia; lymphoma}}, language = {{eng}}, number = {{4}}, publisher = {{MDPI AG}}, series = {{Pharmaceutics}}, title = {{New and Effective Inhibitor of Class I HDACs, Eimbinostat, Reduces the Growth of Hematologic Cancer Cells and Triggers Apoptosis}}, url = {{http://dx.doi.org/10.3390/pharmaceutics17040416}}, doi = {{10.3390/pharmaceutics17040416}}, volume = {{17}}, year = {{2025}}, }