Aortic Stiffness, Inflammation, and Incidence of Cardiovascular Events in Elderly Participants From the General Population
(2022) In Angiology 73(1). p.51-59- Abstract
Low-grade inflammation and arterial stiffness are key factors in the development of vascular aging. However, the interplay between arterial stiffness and inflammation for cardiovascular (CV) disease is unclear. Aortic pulse wave velocity (aPWV) and the inflammatory markers, high-sensitivity C-reactive protein (CRP) and orosomucoid, were measured in 2710 participants (median age: 72 years). These participants were followed up for a mean of 7.6 years for a composite CV disease end point. Per 1 interquartile range increment of CRP and orosomucoid, respectively, aPWV increased by 0.19 m/s (95% CI: 0.07-0.32) and 0.19 m/s (0.11-0.27), after multifactorial adjustment. Mediation analysis showed that aPWV, after multifactorial adjustment,... (More)
Low-grade inflammation and arterial stiffness are key factors in the development of vascular aging. However, the interplay between arterial stiffness and inflammation for cardiovascular (CV) disease is unclear. Aortic pulse wave velocity (aPWV) and the inflammatory markers, high-sensitivity C-reactive protein (CRP) and orosomucoid, were measured in 2710 participants (median age: 72 years). These participants were followed up for a mean of 7.6 years for a composite CV disease end point. Per 1 interquartile range increment of CRP and orosomucoid, respectively, aPWV increased by 0.19 m/s (95% CI: 0.07-0.32) and 0.19 m/s (0.11-0.27), after multifactorial adjustment. Mediation analysis showed that aPWV, after multifactorial adjustment, mediated 8% (−4, 20) of the CV disease risk associated with CRP and 8% (−4, 18) of orosomucoid risk. The associated risk increased with combinations of high aPWV and high CRP or orosomucoid. We found no evidence that arterial PWV acted as an important mediator of the relationship between systemic inflammation and CV disease risk in this elderly population. The results instead indicate an additive effect. Our study supports the view that arterial stiffness and chronic inflammation affects CV risk mainly through separate causal pathways.
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- author
- Nilsson Wadström, Benjamin ; Persson, Margaretha LU ; Engström, Gunnar LU and Nilsson, Peter M. LU
- organization
- publishing date
- 2022
- type
- Contribution to journal
- publication status
- published
- subject
- keywords
- aorta, arterial stiffness, arteriosclerosis, C-reactive protein, inflammation, orosomucoid, pulse wave velocity
- in
- Angiology
- volume
- 73
- issue
- 1
- pages
- 51 - 59
- publisher
- SAGE Publications
- external identifiers
-
- scopus:85106436105
- pmid:34013787
- ISSN
- 0003-3197
- DOI
- 10.1177/00033197211017406
- language
- English
- LU publication?
- yes
- id
- f87ee17e-9bff-4577-92ab-2ad107990800
- date added to LUP
- 2021-06-15 15:43:50
- date last changed
- 2024-11-17 05:23:51
@article{f87ee17e-9bff-4577-92ab-2ad107990800, abstract = {{<p>Low-grade inflammation and arterial stiffness are key factors in the development of vascular aging. However, the interplay between arterial stiffness and inflammation for cardiovascular (CV) disease is unclear. Aortic pulse wave velocity (aPWV) and the inflammatory markers, high-sensitivity C-reactive protein (CRP) and orosomucoid, were measured in 2710 participants (median age: 72 years). These participants were followed up for a mean of 7.6 years for a composite CV disease end point. Per 1 interquartile range increment of CRP and orosomucoid, respectively, aPWV increased by 0.19 m/s (95% CI: 0.07-0.32) and 0.19 m/s (0.11-0.27), after multifactorial adjustment. Mediation analysis showed that aPWV, after multifactorial adjustment, mediated 8% (−4, 20) of the CV disease risk associated with CRP and 8% (−4, 18) of orosomucoid risk. The associated risk increased with combinations of high aPWV and high CRP or orosomucoid. We found no evidence that arterial PWV acted as an important mediator of the relationship between systemic inflammation and CV disease risk in this elderly population. The results instead indicate an additive effect. Our study supports the view that arterial stiffness and chronic inflammation affects CV risk mainly through separate causal pathways.</p>}}, author = {{Nilsson Wadström, Benjamin and Persson, Margaretha and Engström, Gunnar and Nilsson, Peter M.}}, issn = {{0003-3197}}, keywords = {{aorta; arterial stiffness; arteriosclerosis; C-reactive protein; inflammation; orosomucoid; pulse wave velocity}}, language = {{eng}}, number = {{1}}, pages = {{51--59}}, publisher = {{SAGE Publications}}, series = {{Angiology}}, title = {{Aortic Stiffness, Inflammation, and Incidence of Cardiovascular Events in Elderly Participants From the General Population}}, url = {{http://dx.doi.org/10.1177/00033197211017406}}, doi = {{10.1177/00033197211017406}}, volume = {{73}}, year = {{2022}}, }