Heiko Herwald
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- 2003
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Mark
Contact activation by pathogenic bacteria: a virulence mechanism contributing to the pathophysiology of sepsis.
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- Contribution to journal › Article
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Mark
Superantigens from Staphylococcus aureus induce procoagulant activity and monocyte tissue factor expression in whole blood and mononuclear cells via IL-1 beta
(
- Contribution to journal › Article
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Mark
The conversion of fibrinogen to fibrin at the surface of curliated E. coli bacteria leads to the generation of proinflammatory fibrinopeptides.
(
- Contribution to journal › Article
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Mark
Superantigens from Staphylococcus aureus induce procoagulant activity and monocyte tissue factor expression in whole blood and mononuclear cells via IL-1beta.
(
- Contribution to journal › Article
- 2002
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Mark
Contact-system activation in children with vasculitis.
(
- Contribution to journal › Article
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Mark
Secretion of heparin-binding protein from human neutrophils is determined by its localization in azurophilic granules and secretory vesicles.
(
- Contribution to journal › Article
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Mark
Identification of two protein-binding and functional regions of curli, a surface organelle and virulence determinant of Escherichia coli
(
- Contribution to journal › Article
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Mark
Peptidoglycan from Staphylococcus aureus induces tissue factor expression and procoagulant activity in human monocytes.
(
- Contribution to journal › Article
- 2001
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Mark
Heparin-binding protein (HBP/CAP37): A missing link in neutrophil-evoked alteration of vascular permeability
(
- Contribution to journal › Article
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Mark
Zinc-dependent conformational changes in domain D5 of high molecular mass kininogen modulate contact activation
(
- Contribution to journal › Article