Genetic variation in the gene encoding calpain-10 is associated with type 2 diabetes mellitus
(2000) In Nature Genetics 26(2). p.163-175- Abstract
- Type 2 or non-insulin-dependent diabetes mellitus (NIDDM) is the most common form of diabetes worldwide, affecting approximately 4% of the world's adult population. It is multifactorial in origin with both genetic and environmental factors contributing to its development. A genome-wide screen for type 2 diabetes genes carried out in Mexican Americans localized a susceptibility gene, designated NIDDM1, to chromosome 2. Here we describe the positional cloning of a gene located in the NIDDM1 region that shows association with type 2 diabetes in Mexican Americans and a Northern European population from the Botnia region of Finland. This putative diabetes-susceptibility gene encodes a ubiquitously expressed member of the calpain-like cysteine... (More)
- Type 2 or non-insulin-dependent diabetes mellitus (NIDDM) is the most common form of diabetes worldwide, affecting approximately 4% of the world's adult population. It is multifactorial in origin with both genetic and environmental factors contributing to its development. A genome-wide screen for type 2 diabetes genes carried out in Mexican Americans localized a susceptibility gene, designated NIDDM1, to chromosome 2. Here we describe the positional cloning of a gene located in the NIDDM1 region that shows association with type 2 diabetes in Mexican Americans and a Northern European population from the Botnia region of Finland. This putative diabetes-susceptibility gene encodes a ubiquitously expressed member of the calpain-like cysteine protease family, calpain-10 (CAPN10). This finding suggests a novel pathway that may contribute to the development of type 2 diabetes. (Less)
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https://lup.lub.lu.se/record/1118025
- author
- organization
- publishing date
- 2000
- type
- Contribution to journal
- publication status
- published
- subject
- in
- Nature Genetics
- volume
- 26
- issue
- 2
- pages
- 163 - 175
- publisher
- Nature Publishing Group
- external identifiers
-
- pmid:11017071
- scopus:0033772073
- pmid:11017071
- ISSN
- 1546-1718
- DOI
- 10.1038/79876
- language
- English
- LU publication?
- yes
- id
- ad4d5ab3-7a8c-41d2-86b9-c740b0fa2a81 (old id 1118025)
- date added to LUP
- 2016-04-01 16:44:12
- date last changed
- 2024-05-11 06:46:42
@article{ad4d5ab3-7a8c-41d2-86b9-c740b0fa2a81, abstract = {{Type 2 or non-insulin-dependent diabetes mellitus (NIDDM) is the most common form of diabetes worldwide, affecting approximately 4% of the world's adult population. It is multifactorial in origin with both genetic and environmental factors contributing to its development. A genome-wide screen for type 2 diabetes genes carried out in Mexican Americans localized a susceptibility gene, designated NIDDM1, to chromosome 2. Here we describe the positional cloning of a gene located in the NIDDM1 region that shows association with type 2 diabetes in Mexican Americans and a Northern European population from the Botnia region of Finland. This putative diabetes-susceptibility gene encodes a ubiquitously expressed member of the calpain-like cysteine protease family, calpain-10 (CAPN10). This finding suggests a novel pathway that may contribute to the development of type 2 diabetes.}}, author = {{Horikawa, Y and Oda, N and Cox, N J and Li, X and Orho-Melander, Marju and Hara, M and Hinokio, Y and Lindner, T H and Mashima, H and Schwarz, P E and del Bosque-Plata, L and Horikawa, Y and Oda, Y and Yoshiuchi, I and Colilla, S and Polonsky, K S and Wei, S and Concannon, P and Iwasaki, N and Schulze, J and Baier, Leslie J. and Bogardus, Clifton and Groop, Leif and Boerwinkle, Eric and Hanis, Craig L. and Bell, Graeme I.}}, issn = {{1546-1718}}, language = {{eng}}, number = {{2}}, pages = {{163--175}}, publisher = {{Nature Publishing Group}}, series = {{Nature Genetics}}, title = {{Genetic variation in the gene encoding calpain-10 is associated with type 2 diabetes mellitus}}, url = {{http://dx.doi.org/10.1038/79876}}, doi = {{10.1038/79876}}, volume = {{26}}, year = {{2000}}, }