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Angiogenesis is increased in advanced haemophilic joint disease and characterized by normal pericyte coverage.

Zetterberg, Eva LU ; Palmblad, J; Wallensten, R; Morfini, M; Melchiorre, D and Holmström, Eva M LU (2014) In European Journal of Haematology 92(3). p.256-262
Abstract
Repeated intra-articular bleedings in patients with haemophilia results in a crippling arthropathy for which no specific treatment is currently available. Recent studies have shown that neoangiogenesis is involved in the pathologic process. The aim of this study was to determine if angiogenesis is dysregulated in haemophilic joint disease (HJD).
Please use this url to cite or link to this publication:
author
organization
publishing date
type
Contribution to journal
publication status
published
subject
in
European Journal of Haematology
volume
92
issue
3
pages
256 - 262
publisher
Wiley-Blackwell
external identifiers
  • pmid:24168433
  • wos:000331458500011
  • scopus:84894227413
ISSN
1600-0609
DOI
10.1111/ejh.12227
language
English
LU publication?
yes
id
0b14a2e9-3278-4279-a92a-7586a64e2011 (old id 4142891)
alternative location
http://www.ncbi.nlm.nih.gov/pubmed/24168433?dopt=Abstract
date added to LUP
2013-11-04 21:24:08
date last changed
2017-01-08 03:16:09
@article{0b14a2e9-3278-4279-a92a-7586a64e2011,
  abstract     = {Repeated intra-articular bleedings in patients with haemophilia results in a crippling arthropathy for which no specific treatment is currently available. Recent studies have shown that neoangiogenesis is involved in the pathologic process. The aim of this study was to determine if angiogenesis is dysregulated in haemophilic joint disease (HJD).},
  author       = {Zetterberg, Eva and Palmblad, J and Wallensten, R and Morfini, M and Melchiorre, D and Holmström, Eva M},
  issn         = {1600-0609},
  language     = {eng},
  number       = {3},
  pages        = {256--262},
  publisher    = {Wiley-Blackwell},
  series       = {European Journal of Haematology},
  title        = {Angiogenesis is increased in advanced haemophilic joint disease and characterized by normal pericyte coverage.},
  url          = {http://dx.doi.org/10.1111/ejh.12227},
  volume       = {92},
  year         = {2014},
}