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Deciphering high density lipoprotein (HDL) structure-function : Detailed analysis of HDL subfractions reveals molecular differences leading to atherosclerosis risk

Correa, Yubexi ; Felderer, Birgit ; Jansen, Martin ; Gil-Carton, David ; Blanchet, Clement ; Roosen-Runge, Felix LU ; Pedersen, Jan Skov and Cárdenas, Marité LU (2026) In International Journal of Biological Macromolecules 363.
Abstract

High-density lipoproteins (HDL) are crucial for cardiovascular health. HDL dysfunction is strongly linked to atherosclerosis and other diseases. Although HDL is often treated as a single entity, it comprises a spectrum of subfractions differing in size, structure, and composition. Understanding the ultrastructure and function of these subfractions is essential for uncovering the molecular mechanisms behind HDL dysfunction and improving disease risk prediction. We employed Small-Angle X-ray Scattering (SAXS) and cryogenic-electron tomography (cryo-ET) to analyze the structural features of total HDL and its subfractions: HDL2b, HDL2a, and HDL3. We present updated, detailed structural models showing that... (More)

High-density lipoproteins (HDL) are crucial for cardiovascular health. HDL dysfunction is strongly linked to atherosclerosis and other diseases. Although HDL is often treated as a single entity, it comprises a spectrum of subfractions differing in size, structure, and composition. Understanding the ultrastructure and function of these subfractions is essential for uncovering the molecular mechanisms behind HDL dysfunction and improving disease risk prediction. We employed Small-Angle X-ray Scattering (SAXS) and cryogenic-electron tomography (cryo-ET) to analyze the structural features of total HDL and its subfractions: HDL2b, HDL2a, and HDL3. We present updated, detailed structural models showing that the structure of HDL changes dramatically from the smallest to the largest subfraction, with HDL3 partially exposing the core to the particle's surface. Moreover, total HDL reflects the weighted sum of its subfractions, emphasizing that analysis of total HDL alone may be misleading. HDL subfractions were also studied in serum samples from 16 individuals classified as low- or high-risk for atherosclerosis. Even though biochemical differences appeared mainly in HDL2, structural differences were most pronounced in HDL3 addressing that particle composition alone cannot fully distinguish HDL dysfunction. Multimodal analysis integrating structural and biochemical data was able to separate risk groups and revealed correlations between structural parameters and cardiovascular risk status. Our findings support the importance of analyzing HDL subfractions to uncover molecular drivers of disease risk and suggest candidate biomarkers for atherosclerosis upon validation in larger cohorts.

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author
; ; ; ; ; ; and
organization
publishing date
type
Contribution to journal
publication status
published
subject
keywords
Biomedicine, Diagnostics and therapy, High-density lipoprotein, Lipid serum profile, Small-angle X-ray scattering
in
International Journal of Biological Macromolecules
volume
363
article number
152016
publisher
Elsevier
external identifiers
  • scopus:105036689908
  • pmid:41991129
ISSN
0141-8130
DOI
10.1016/j.ijbiomac.2026.152016
language
English
LU publication?
yes
id
471b206e-906b-48f3-a2c6-3ed50600f810
date added to LUP
2026-05-29 14:32:32
date last changed
2026-05-29 14:33:06
@article{471b206e-906b-48f3-a2c6-3ed50600f810,
  abstract     = {{<p>High-density lipoproteins (HDL) are crucial for cardiovascular health. HDL dysfunction is strongly linked to atherosclerosis and other diseases. Although HDL is often treated as a single entity, it comprises a spectrum of subfractions differing in size, structure, and composition. Understanding the ultrastructure and function of these subfractions is essential for uncovering the molecular mechanisms behind HDL dysfunction and improving disease risk prediction. We employed Small-Angle X-ray Scattering (SAXS) and cryogenic-electron tomography (cryo-ET) to analyze the structural features of total HDL and its subfractions: HDL<sub>2b</sub>, HDL<sub>2a</sub>, and HDL<sub>3</sub>. We present updated, detailed structural models showing that the structure of HDL changes dramatically from the smallest to the largest subfraction, with HDL<sub>3</sub> partially exposing the core to the particle's surface. Moreover, total HDL reflects the weighted sum of its subfractions, emphasizing that analysis of total HDL alone may be misleading. HDL subfractions were also studied in serum samples from 16 individuals classified as low- or high-risk for atherosclerosis. Even though biochemical differences appeared mainly in HDL<sub>2</sub>, structural differences were most pronounced in HDL<sub>3</sub> addressing that particle composition alone cannot fully distinguish HDL dysfunction. Multimodal analysis integrating structural and biochemical data was able to separate risk groups and revealed correlations between structural parameters and cardiovascular risk status. Our findings support the importance of analyzing HDL subfractions to uncover molecular drivers of disease risk and suggest candidate biomarkers for atherosclerosis upon validation in larger cohorts.</p>}},
  author       = {{Correa, Yubexi and Felderer, Birgit and Jansen, Martin and Gil-Carton, David and Blanchet, Clement and Roosen-Runge, Felix and Pedersen, Jan Skov and Cárdenas, Marité}},
  issn         = {{0141-8130}},
  keywords     = {{Biomedicine; Diagnostics and therapy; High-density lipoprotein; Lipid serum profile; Small-angle X-ray scattering}},
  language     = {{eng}},
  publisher    = {{Elsevier}},
  series       = {{International Journal of Biological Macromolecules}},
  title        = {{Deciphering high density lipoprotein (HDL) structure-function : Detailed analysis of HDL subfractions reveals molecular differences leading to atherosclerosis risk}},
  url          = {{http://dx.doi.org/10.1016/j.ijbiomac.2026.152016}},
  doi          = {{10.1016/j.ijbiomac.2026.152016}},
  volume       = {{363}},
  year         = {{2026}},
}