Early diagnostic value of Bcl-3 localization in colorectal cancer.
Saamarthy, Karunakar LU ; Björner, Sofie LU ; Johansson, Martin LU ; Landberg, Göran LU ; Massoumi, Ramin LU ; Jirström, Karin LU
and Masoumi, Katarzyna
LU
(2015)
In BMC Cancer
15(1).
- Abstract
- B-cell leukemia 3 (Bcl-3) is a member of the inhibitor of κB family, which regulates a wide range of biological processes by functioning as a transcriptional activator or as a repressor of target genes. Elevated expression, sustained nuclear accumulation, and uncontrolled activation of Bcl-3 causes increased cellular proliferation or survival, dependent on the tissue and type of stimuli.
Please use this url to cite or link to this publication:
https://lup.lub.lu.se/record/5461447
- author
- Saamarthy, Karunakar
LU
; Björner, Sofie
LU
; Johansson, Martin
LU
; Landberg, Göran
LU
; Massoumi, Ramin
LU
; Jirström, Karin
LU
and Masoumi, Katarzyna
LU
- organization
- publishing date
- 2015
- type
- Contribution to journal
- publication status
- published
- subject
- in
- BMC Cancer
- volume
- 15
- issue
- 1
- article number
- 341
- publisher
- BioMed Central (BMC)
- external identifiers
-
- pmid:25929479
- wos:000354837400001
- pmid:25929479
- scopus:85019178863
- ISSN
- 1471-2407
- DOI
- 10.1186/s12885-015-1342-6
- language
- English
- LU publication?
- yes
- additional info
- The information about affiliations in this record was updated in December 2015. The record was previously connected to the following departments: Division of Translational Cancer Research (013250500), Pathology (Malmö) (013031000), Clinical pathology, Malmö (013017551), Pathology, (Lund) (013030000)
- id
- 757e90a6-5f6a-4a38-a7ba-835f4ad35ea6 (old id 5461447)
- alternative location
- http://www.ncbi.nlm.nih.gov/pubmed/25929479?dopt=Abstract
- date added to LUP
- 2016-04-01 13:01:18
- date last changed
- 2024-01-24 04:19:37
@article{757e90a6-5f6a-4a38-a7ba-835f4ad35ea6,
abstract = {{B-cell leukemia 3 (Bcl-3) is a member of the inhibitor of κB family, which regulates a wide range of biological processes by functioning as a transcriptional activator or as a repressor of target genes. Elevated expression, sustained nuclear accumulation, and uncontrolled activation of Bcl-3 causes increased cellular proliferation or survival, dependent on the tissue and type of stimuli.}},
author = {{Saamarthy, Karunakar and Björner, Sofie and Johansson, Martin and Landberg, Göran and Massoumi, Ramin and Jirström, Karin and Masoumi, Katarzyna}},
issn = {{1471-2407}},
language = {{eng}},
number = {{1}},
publisher = {{BioMed Central (BMC)}},
series = {{BMC Cancer}},
title = {{Early diagnostic value of Bcl-3 localization in colorectal cancer.}},
url = {{https://lup.lub.lu.se/search/files/3110264/8411659.pdf}},
doi = {{10.1186/s12885-015-1342-6}},
volume = {{15}},
year = {{2015}},
}