Regulation of ADP-ribosyltransferase activity by ART domain dimerization in PARP15
(2025) In Nature Communications 16.- Abstract
PARP15 is a mono-ADP-ribosyltransferase that targets an unknown set of proteins as well as RNA. Its evolutionary relationship with PARP14 suggests roles in antiviral defence; its localization to stress granules points to functions in the regulation of translation. Here we show that the transferase domain of PARP15 dimerizes in solution; the formation of dimers is a prerequisite for catalytic activity and monomeric mutant variants of the domain are inactive. In cells, dimer-disrupting mutations abrogate catalytic activity and alter the subcellular localization of the full-length protein. Using biophysical methods, including X-ray crystallography and HDX-MS, we provide evidence for a regulatory mechanism by which dimerization enables... (More)
PARP15 is a mono-ADP-ribosyltransferase that targets an unknown set of proteins as well as RNA. Its evolutionary relationship with PARP14 suggests roles in antiviral defence; its localization to stress granules points to functions in the regulation of translation. Here we show that the transferase domain of PARP15 dimerizes in solution; the formation of dimers is a prerequisite for catalytic activity and monomeric mutant variants of the domain are inactive. In cells, dimer-disrupting mutations abrogate catalytic activity and alter the subcellular localization of the full-length protein. Using biophysical methods, including X-ray crystallography and HDX-MS, we provide evidence for a regulatory mechanism by which dimerization enables correct target engagement rather than NAD+ co-substrate binding, and by which the two protomers of the dimer operate independently of one another. Together, our results uncover a regulatory mechanism in a PARP family enzyme.
(Less)
- author
- Ebenwaldner, Carmen
LU
; García Saura, Antonio Ginés
LU
; Ekström, Simon
LU
; Bernfur, Katja
LU
; Moche, Martin
; Logan, Derek T.
LU
; Cohen, Michael S.
and Schüler, Herwig
LU
- organization
- publishing date
- 2025-12
- type
- Contribution to journal
- publication status
- published
- subject
- in
- Nature Communications
- volume
- 16
- article number
- 9567
- pages
- 17 pages
- publisher
- Nature Publishing Group
- external identifiers
-
- scopus:105020278137
- pmid:41162413
- ISSN
- 2041-1723
- DOI
- 10.1038/s41467-025-65315-9
- language
- English
- LU publication?
- yes
- additional info
- Publisher Copyright: © The Author(s) 2025.
- id
- 711bdf09-1e49-4fe6-92ec-8a965db4b69c
- date added to LUP
- 2025-11-10 09:13:01
- date last changed
- 2025-11-14 03:29:32
@article{711bdf09-1e49-4fe6-92ec-8a965db4b69c,
abstract = {{<p>PARP15 is a mono-ADP-ribosyltransferase that targets an unknown set of proteins as well as RNA. Its evolutionary relationship with PARP14 suggests roles in antiviral defence; its localization to stress granules points to functions in the regulation of translation. Here we show that the transferase domain of PARP15 dimerizes in solution; the formation of dimers is a prerequisite for catalytic activity and monomeric mutant variants of the domain are inactive. In cells, dimer-disrupting mutations abrogate catalytic activity and alter the subcellular localization of the full-length protein. Using biophysical methods, including X-ray crystallography and HDX-MS, we provide evidence for a regulatory mechanism by which dimerization enables correct target engagement rather than NAD<sup>+</sup> co-substrate binding, and by which the two protomers of the dimer operate independently of one another. Together, our results uncover a regulatory mechanism in a PARP family enzyme.</p>}},
author = {{Ebenwaldner, Carmen and García Saura, Antonio Ginés and Ekström, Simon and Bernfur, Katja and Moche, Martin and Logan, Derek T. and Cohen, Michael S. and Schüler, Herwig}},
issn = {{2041-1723}},
language = {{eng}},
publisher = {{Nature Publishing Group}},
series = {{Nature Communications}},
title = {{Regulation of ADP-ribosyltransferase activity by ART domain dimerization in PARP15}},
url = {{http://dx.doi.org/10.1038/s41467-025-65315-9}},
doi = {{10.1038/s41467-025-65315-9}},
volume = {{16}},
year = {{2025}},
}