Advanced

Hypertensive encephalopathy and cerebral infarction

Edvardsson, Bengt LU (2014) In SpringerPlus 3.
Abstract
Introduction: Hypertensive encephalopathy is one cause of posterior reversible encephalopathy syndrome. Hypertensive encephalopathy and cerebral infarction have only been reported in a few individual case reports. Case description: A 51-year-old woman presented with hypertensive encephalopathy. T2-weighted images from magnetic resonance imaging showed hyperintense lesions in both occipital and parietal lobes. Diffusion-weighted imaging showed that this represented cytotoxic oedema and perfusion magnetic resonance imaging revealed reduced blood volume and flow. The magnetic resonance imaging was repeated 5 months later and subtotal regression of the T2-hyperintensity had occurred. However, small bilateral infarcts were seen on T1-weighted... (More)
Introduction: Hypertensive encephalopathy is one cause of posterior reversible encephalopathy syndrome. Hypertensive encephalopathy and cerebral infarction have only been reported in a few individual case reports. Case description: A 51-year-old woman presented with hypertensive encephalopathy. T2-weighted images from magnetic resonance imaging showed hyperintense lesions in both occipital and parietal lobes. Diffusion-weighted imaging showed that this represented cytotoxic oedema and perfusion magnetic resonance imaging revealed reduced blood volume and flow. The magnetic resonance imaging was repeated 5 months later and subtotal regression of the T2-hyperintensity had occurred. However, small bilateral infarcts were seen on T1-weighted images. Perfusion magnetic resonance imaging presented reduced blood volume and flow on the right side. Discussion and evaluation: The patient in this report had posterior reversible encephalopathy syndrome caused by hypertensive encephalopathy. Magnetic resonance imaging of the brain showed bilateral cytotoxic oedema that partially resolved and resulted in small infarcts. The imaging findings are compatible with posterior reversible encephalopathy syndrome with subtotal resolution and infarct evolution. Conclusion: The case report suggests that the presence of hypertensive encephalopathy and posterior reversible encephalopathy syndrome should alert clinicians and lead to prompt treatment in order to prevent cerebral damage. (Less)
Please use this url to cite or link to this publication:
author
organization
publishing date
type
Contribution to journal
publication status
published
subject
keywords
Hypertensive encephalopathy, Posterior reversible encephalopathy, syndrome, Magnetic resonance imaging, Cerebral infarction, Ischemia
in
SpringerPlus
volume
3
publisher
Springer
external identifiers
  • wos:000359123200001
  • scopus:84928689473
ISSN
2193-1801
DOI
10.1186/2193-1801-3-741
language
English
LU publication?
yes
id
e7b79521-a5cc-473a-a511-ec07aa98b8e1 (old id 7985055)
date added to LUP
2015-10-01 07:28:59
date last changed
2017-01-01 06:10:06
@article{e7b79521-a5cc-473a-a511-ec07aa98b8e1,
  abstract     = {Introduction: Hypertensive encephalopathy is one cause of posterior reversible encephalopathy syndrome. Hypertensive encephalopathy and cerebral infarction have only been reported in a few individual case reports. Case description: A 51-year-old woman presented with hypertensive encephalopathy. T2-weighted images from magnetic resonance imaging showed hyperintense lesions in both occipital and parietal lobes. Diffusion-weighted imaging showed that this represented cytotoxic oedema and perfusion magnetic resonance imaging revealed reduced blood volume and flow. The magnetic resonance imaging was repeated 5 months later and subtotal regression of the T2-hyperintensity had occurred. However, small bilateral infarcts were seen on T1-weighted images. Perfusion magnetic resonance imaging presented reduced blood volume and flow on the right side. Discussion and evaluation: The patient in this report had posterior reversible encephalopathy syndrome caused by hypertensive encephalopathy. Magnetic resonance imaging of the brain showed bilateral cytotoxic oedema that partially resolved and resulted in small infarcts. The imaging findings are compatible with posterior reversible encephalopathy syndrome with subtotal resolution and infarct evolution. Conclusion: The case report suggests that the presence of hypertensive encephalopathy and posterior reversible encephalopathy syndrome should alert clinicians and lead to prompt treatment in order to prevent cerebral damage.},
  articleno    = {741},
  author       = {Edvardsson, Bengt},
  issn         = {2193-1801},
  keyword      = {Hypertensive encephalopathy,Posterior reversible encephalopathy,syndrome,Magnetic resonance imaging,Cerebral infarction,Ischemia},
  language     = {eng},
  publisher    = {Springer},
  series       = {SpringerPlus},
  title        = {Hypertensive encephalopathy and cerebral infarction},
  url          = {http://dx.doi.org/10.1186/2193-1801-3-741},
  volume       = {3},
  year         = {2014},
}