Polystyrene Nanoplastic Exposure Promotes Amyloid Misfolding and Metabolic Impairment at Sub-Lethal Doses. A Subcellular Infrared Imaging Study
(2026) In Environmental Science: Nano 13(4). p.1948-1961- Abstract
- Microplastics and nanoplastics (MNPs) are ubiquitous environmental pollutants with increasing implications for human health. While their presence in human tissues is established, the molecular mechanisms driving their potential neurotoxicity remain unclear. This study investigates the impact of polystyrene (PS) on amyloid protein misfolding and cellular metabolism using Optical Photothermal Infrared (O-PTIR) spectroscopy, a label-free, sub-diffraction imaging technique. Our results reveal that PS exposure promotes pathological protein misfolding, specifically decreasing β-sheet-rich conformations, and disrupts metabolic homeostasis at sub-lethal doses. These suggest that the nanoplastic surface acts as a catalytic scaffold for amyloid... (More)
- Microplastics and nanoplastics (MNPs) are ubiquitous environmental pollutants with increasing implications for human health. While their presence in human tissues is established, the molecular mechanisms driving their potential neurotoxicity remain unclear. This study investigates the impact of polystyrene (PS) on amyloid protein misfolding and cellular metabolism using Optical Photothermal Infrared (O-PTIR) spectroscopy, a label-free, sub-diffraction imaging technique. Our results reveal that PS exposure promotes pathological protein misfolding, specifically decreasing β-sheet-rich conformations, and disrupts metabolic homeostasis at sub-lethal doses. These suggest that the nanoplastic surface acts as a catalytic scaffold for amyloid aggregation, driving cellular dysfunction prior to acute toxicity. This identifies a plausible molecular pathway by which environmental MNP pollution contributes to the risk and progression of neurodegenerative diseases, highlighting the need for risk assessments that look beyond simple cell survival. (Less)
Please use this url to cite or link to this publication:
https://lup.lub.lu.se/record/d5e7c491-f9b1-43c1-8d87-bfa78bb07863
- author
- Augusto Silva, Iran
LU
; Paulus, Agnes
LU
; Skoryk, Valeriia
LU
; Kar Yan, Su
LU
; Herranz-Trillo, Fátima
LU
and KLEMENTIEVA, Oxana
LU
- organization
-
- Medical Microspectroscopy (research group)
- MAX IV, Science division
- MultiPark: Multidisciplinary research on neurodegenerative diseases
- LU Profile Area: Light and Materials
- LTH Profile Area: Nanoscience and Semiconductor Technology
- NanoLund: Centre for Nanoscience
- Stem Cell Center
- WCMM-Wallenberg Centre for Molecular Medicine
- MAX IV Laboratory
- Lung Bioengineering and Regeneration (research group)
- Infect@LU
- LU Profile Area: Proactive Ageing
- publishing date
- 2026
- type
- Contribution to journal
- publication status
- published
- subject
- in
- Environmental Science: Nano
- volume
- 13
- issue
- 4
- pages
- 1948 - 1961
- publisher
- Royal Society of Chemistry
- external identifiers
-
- scopus:105033825413
- ISSN
- 2051-8153
- DOI
- 10.1039/D5EN01181G
- language
- English
- LU publication?
- yes
- id
- d5e7c491-f9b1-43c1-8d87-bfa78bb07863
- date added to LUP
- 2026-03-10 21:58:28
- date last changed
- 2026-05-13 08:17:19
@article{d5e7c491-f9b1-43c1-8d87-bfa78bb07863,
abstract = {{Microplastics and nanoplastics (MNPs) are ubiquitous environmental pollutants with increasing implications for human health. While their presence in human tissues is established, the molecular mechanisms driving their potential neurotoxicity remain unclear. This study investigates the impact of polystyrene (PS) on amyloid protein misfolding and cellular metabolism using Optical Photothermal Infrared (O-PTIR) spectroscopy, a label-free, sub-diffraction imaging technique. Our results reveal that PS exposure promotes pathological protein misfolding, specifically decreasing β-sheet-rich conformations, and disrupts metabolic homeostasis at sub-lethal doses. These suggest that the nanoplastic surface acts as a catalytic scaffold for amyloid aggregation, driving cellular dysfunction prior to acute toxicity. This identifies a plausible molecular pathway by which environmental MNP pollution contributes to the risk and progression of neurodegenerative diseases, highlighting the need for risk assessments that look beyond simple cell survival.}},
author = {{Augusto Silva, Iran and Paulus, Agnes and Skoryk, Valeriia and Kar Yan, Su and Herranz-Trillo, Fátima and KLEMENTIEVA, Oxana}},
issn = {{2051-8153}},
language = {{eng}},
number = {{4}},
pages = {{1948--1961}},
publisher = {{Royal Society of Chemistry}},
series = {{Environmental Science: Nano}},
title = {{Polystyrene Nanoplastic Exposure Promotes Amyloid Misfolding and Metabolic Impairment at Sub-Lethal Doses. A Subcellular Infrared Imaging Study}},
url = {{http://dx.doi.org/10.1039/D5EN01181G}},
doi = {{10.1039/D5EN01181G}},
volume = {{13}},
year = {{2026}},
}