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Improving lymphopoiesis in aged bone marrow

Konturek-Ciesla, Anna LU and Bryder, David LU (2026) In Current Opinion in Hematology 33(4).
Abstract

Purpose of review – Aging is associated with impaired B lymphopoiesis and T lymphopoiesis, contributing to immunosenescence and poor immune recovery. Although this decline can be attributed to intrinsic hematopoietic stem cell aging, growing evidence indicates that lymphoid failure reflects constraints operating across multiple levels of the hematopoietic system. This review frames age-associated lymphopoiesis decline as a systems-level problem and outlines conceptual avenues for therapeutic intervention.Recent findings – Age-associated lymphoid failure is increasingly attributed to inflammatory suppression, dominance of dysfunctional stem and progenitor states, and compromised extramedullary support. These insights provide a framework... (More)

Purpose of review – Aging is associated with impaired B lymphopoiesis and T lymphopoiesis, contributing to immunosenescence and poor immune recovery. Although this decline can be attributed to intrinsic hematopoietic stem cell aging, growing evidence indicates that lymphoid failure reflects constraints operating across multiple levels of the hematopoietic system. This review frames age-associated lymphopoiesis decline as a systems-level problem and outlines conceptual avenues for therapeutic intervention.Recent findings – Age-associated lymphoid failure is increasingly attributed to inflammatory suppression, dominance of dysfunctional stem and progenitor states, and compromised extramedullary support. These insights provide a framework for interventions that restore immune competence by rebalancing hematopoiesis or selectively replacing compromised stem cell function.Summary – Age-associated lymphoid decline arises from coordinated constraints across the bone marrow niche, stem and progenitor composition, and extramedullary lymphoid support, rather than intrinsic stem cell exhaustion alone. Targeting these bottlenecks in a context-dependent manner offers multiple routes to improve lymphopoiesis and restore immune competence in aging.

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author
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organization
publishing date
type
Contribution to journal
publication status
published
subject
keywords
hematopoietic stem and progenitor cells, immune aging, lymphopoiesis, regeneration
in
Current Opinion in Hematology
volume
33
issue
4
publisher
Lippincott Williams & Wilkins
external identifiers
  • pmid:41861249
  • scopus:105037559697
ISSN
1065-6251
DOI
10.1097/MOH.0000000000000923
language
English
LU publication?
yes
additional info
Publisher Copyright: Copyright © 2026 The Author(s). Published by Wolters Kluwer Health, Inc.
id
e1d5f4a1-e71e-4b0d-8f3e-99677338c4e0
date added to LUP
2026-07-17 12:47:08
date last changed
2026-07-18 03:00:02
@article{e1d5f4a1-e71e-4b0d-8f3e-99677338c4e0,
  abstract     = {{<p>Purpose of review – Aging is associated with impaired B lymphopoiesis and T lymphopoiesis, contributing to immunosenescence and poor immune recovery. Although this decline can be attributed to intrinsic hematopoietic stem cell aging, growing evidence indicates that lymphoid failure reflects constraints operating across multiple levels of the hematopoietic system. This review frames age-associated lymphopoiesis decline as a systems-level problem and outlines conceptual avenues for therapeutic intervention.Recent findings – Age-associated lymphoid failure is increasingly attributed to inflammatory suppression, dominance of dysfunctional stem and progenitor states, and compromised extramedullary support. These insights provide a framework for interventions that restore immune competence by rebalancing hematopoiesis or selectively replacing compromised stem cell function.Summary – Age-associated lymphoid decline arises from coordinated constraints across the bone marrow niche, stem and progenitor composition, and extramedullary lymphoid support, rather than intrinsic stem cell exhaustion alone. Targeting these bottlenecks in a context-dependent manner offers multiple routes to improve lymphopoiesis and restore immune competence in aging.</p>}},
  author       = {{Konturek-Ciesla, Anna and Bryder, David}},
  issn         = {{1065-6251}},
  keywords     = {{hematopoietic stem and progenitor cells; immune aging; lymphopoiesis; regeneration}},
  language     = {{eng}},
  number       = {{4}},
  publisher    = {{Lippincott Williams & Wilkins}},
  series       = {{Current Opinion in Hematology}},
  title        = {{Improving lymphopoiesis in aged bone marrow}},
  url          = {{http://dx.doi.org/10.1097/MOH.0000000000000923}},
  doi          = {{10.1097/MOH.0000000000000923}},
  volume       = {{33}},
  year         = {{2026}},
}