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- 2010
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Mark
Recombinant human complement component C2 produced in a human cell line restores the classical complement pathway activity in-vitro: an alternative treatment for C2 deficiency diseases
(
- Contribution to journal › Article
-
Mark
Platelet transcriptional profile and protein expression in patients with systemic lupus erythematosus: up-regulation of the type I interferon system is strongly associated with vascular disease.
(
- Contribution to journal › Article
- 2009
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Mark
C1q Inhibits Immune Complex-Induced Interferon-alpha Production in Plasmacytoid Dendritic Cells A Novel Link Between C1q Deficiency and Systemic Lupus Erythematosus Pathogenesis
(
- Contribution to journal › Article
-
Mark
C1q inhibits immune complex induced IFN-alpha production in plasmacytoid dendritic cells-A novel link between C1q deficiency and SLE pathogenesis
2009) 12th European Meeting on Complement in Human Disease In Molecular Immunology 46(14). p.2857-2857(
- Contribution to journal › Published meeting abstract
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Mark
Complement classical pathway components are all important in clearance of apoptotic and secondary necrotic cells.
(
- Contribution to journal › Article
- 2008
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Mark
SLE serum induces classical caspase-dependent apoptosis independent of death receptors
(
- Contribution to journal › Article
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Mark
Role of C1q in regulation of IFN-alpha production
2008) 72nd Annual Scientific Meeting of the American-College-of-Rheumatology/43rd Annual Scientific Meeting of the Association-of-Rheumatology-Health-Professionals In Arthritis and Rheumatism 58(9). p.706-706(
- Contribution to journal › Published meeting abstract
-
Mark
Complement classical pathway components are all important in clearance of apoptotic cells
(
- Contribution to journal › Published meeting abstract
- 2007
-
Mark
Genetically determined mannan-binding lectin deficiency is of minor importance in determining susceptibility to severe infections and vascular organ damage in systemic lupus erythematosus.
(
- Contribution to journal › Article
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Mark
Association between SLE nephritis and polymorphic variants of the CRP and Fc gamma RIIIa genes
(
- Contribution to journal › Article