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- 2010
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Mark
Overexpression of Alpha2A-Adrenergic Receptors Contributes to Type 2 Diabetes.
(
- Contribution to journal › Article
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Mark
GLP-1 inhibits and adrenaline stimulates glucagon release by differential modulation of N- and L-type Ca2+ channel-dependent exocytosis.
(
- Contribution to journal › Article
- 2009
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Mark
Cell-cell communication between adipocytes and pancreatic beta-cells in acoustically levitated droplets
(
- Contribution to journal › Article
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Mark
Hyperactivity in adrenergic signalling via alpha2A receptors contributes to human type 2 diabetes
2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.108-108(
- Contribution to journal › Published meeting abstract
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Mark
Impaired Insulin Exocytosis in Neural Cell Adhesion Molecule(-/-) Mice Due to Defective Reorganization of the Submembrane F-Actin Network
(
- Contribution to journal › Article
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Mark
Suppression of sulfonylurea- and glucose-induced insulin secretion in vitro and in vivo in mice lacking the chloride transport protein ClC-3.
(
- Contribution to journal › Article
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Mark
Deletion of the G protein-coupled Receptor GPR30 Impairs Glucose Tolerance, Reduces Bone Growth, Increases Blood Pressure, and Eliminates Estradiol-stimulated Insulin Release in Female Mice.
(
- Contribution to journal › Article
- 2008
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Mark
Probability of Exocytosis in Pancreatic β-Cells : Dependence on Ca2+ Sensing Latency Times, Ca2+ Channel Kinetic Parameters, and Channel Clustering
2008) p.299-311(
- Chapter in Book/Report/Conference proceeding › Book chapter
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Mark
Quantification of mRNA in single cells and modelling of RT-qPCR induced noise
(
- Contribution to journal › Article
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Mark
K-ATP-channels and glucagon secretion glucose-regulated
(
- Contribution to journal › Scientific review