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- 2004
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Mark
Collagen type II (CII)-specific antibodies induce arthritis in the absence of T or B cells but the arthritis progression is enhanced by CII-reactive T cells.
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- Contribution to journal › Article
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Mark
A Transient Post-Translationally Modified Form of Cartilage Type II Collagen Is Ignored by Self-Reactive T Cells.
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- Contribution to journal › Article
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Mark
Complement activation by both classical and alternative pathways is critical for the effector phase of arthritis
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- Contribution to journal › Article
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Mark
Enhanced autoimmunity, arthritis, and encephalomyelitis in mice with a reduced oxidative burst due to a mutation in the Ncf1 gene.
(
- Contribution to journal › Article
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Mark
Relapsing polychondritis, induced in mice with matrilin 1, is an antibody- and complement-dependent disease
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- Contribution to journal › Article
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Mark
Analysis of autoreactive T cells associated with murine collagen-induced arthritis using peptide-MHC multimers.
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- Contribution to journal › Article
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Mark
Tracking of proinflammatory collagen-specific T cells in early and late collagen-induced arthritis in humanized mice
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- Contribution to journal › Article
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Mark
Antileukoproteinase - Modulation of neutrophil function and therapeutic effects on anti-type II collagen antibody-induced arthritis
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- Contribution to journal › Article
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Mark
Inconsistent susceptibility to autoimmunity in inbred LEW rats is due to genetic crossbreeding involving segregation of the arthritis-regulating gene Ncf1.
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- Contribution to journal › Article
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Mark
CD1-dependent regulation of chronic central nervous system inflammation in experimental autoimmune encephalomyelitis.
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- Contribution to journal › Article