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- 2001
-
Mark
Gene and Cell-Replacement Therapy in the Treatment of Type 1 Diabetes : How High Must the Standards Be Set?
(
- Contribution to journal › Scientific review
-
Mark
Site-directed mutagenesis of K396R of the 65 kDa glutamic acid decarboxylase active site obliterates enzyme activity but not antibody binding
(
- Contribution to journal › Article
-
Mark
Treatment outcome for IDDM patients in relation to glutamic acid decarboxylase autoantibodies and serum IgG to periodontal pathogens
(
- Contribution to journal › Article
-
Mark
Preservation of enzyme activity and antigenicity after mutagenesis of the membrane anchoring domain of GAD65
(
- Contribution to journal › Article
-
Mark
Prediction of silent celiac disease at diagnosis of childhood type 1 diabetes by tissue transglutaminase autoantibodies and HLA
(
- Contribution to journal › Article
-
Mark
A novel monoclonal antibody specific for the N-terminal end of GAD65
(
- Contribution to journal › Article
-
Mark
Activated human epitope-specific T cells identified by class II tetramers reside within a CD4high, proliferating subset
(
- Contribution to journal › Article
-
Mark
Combinations of beta cell specific autoantibodies at diagnosis of diabetes in young adults reflects different courses of beta cell damage
(
- Contribution to journal › Article
- 2000
-
Mark
Alternative splicing of GAD67 results in the synthesis of a third form of glutamic-acid decarboxylase in human islets and other non-neural tissues
(
- Contribution to journal › Article
-
Mark
The role of glutamic acid decarboxylase and GABA in the pancreas and diabetes.
2000) p.471-484(
- Chapter in Book/Report/Conference proceeding › Book chapter