71 – 80 of 132
- show: 10
- |
- sort: year (new to old)
Close
Embed this list
<iframe src=" "
width=" "
height=" "
allowtransparency="true"
frameborder="0">
</iframe>
- 2009
-
Mark
Imidazoline-induced amplification of glucose- and carbachol-stimulated insulin release includes a marked suppression of islet NO generation in the mouse.
(
- Contribution to journal › Article
-
Mark
The orphan receptor GPR30 and pancreatic islet hormone secretion
2009) 45th Annual Meeting of the European-Association-for-the-Study-of-Diabetes In Diabetologia 52(Suppl 1). p.365-365(
- Contribution to journal › Published meeting abstract
-
Mark
Suppression of sulfonylurea- and glucose-induced insulin secretion in vitro and in vivo in mice lacking the chloride transport protein ClC-3.
(
- Contribution to journal › Article
-
Mark
Glucose Generates Coincident Insulin and Somatostatin Pulses and Antisynchronous Glucagon Pulses from Human Pancreatic Islets.
(
- Contribution to journal › Article
-
Mark
Absence of adenosine A(1) receptors unmasks pulses of insulin release and prolongs those of glucagon and somatostatin.
(
- Contribution to journal › Article
- 2008
-
Mark
GLP-1 inhibits glucagon secretion by affecting several cellular mechanisms
2008) 44th EASD Annual Meeting of the European Association for the Study of Diabetes In Diabetologia 51(S1). p.256-256(
- Contribution to journal › Published meeting abstract
-
Mark
K-ATP-channels and glucagon secretion glucose-regulated
(
- Contribution to journal › Scientific review
-
Mark
Long-term exposure of mouse pancreatic islets to oleate or palmitate results in reduced glucose-induced somatostatin and oversecretion of glucagon
(
- Contribution to journal › Article
-
Mark
Proghrelin-derived peptides influence the secretion of insulin, glucagon, pancreatic polypeptide and somatostatin: A study on isolated islets from mouse and rat pancreas.
(
- Contribution to journal › Article
-
Mark
Rosiglitazone counteracts palmitate-induced beta-cell dysfunction by suppression of MAP kinase, inducible nitric oxide synthase and caspase 3 activities.
(
- Contribution to journal › Article