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- 2007
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Mark
Quantitative Structure-Activity Relationship of Peptides Binding to the Class II Major Histocompatibility Complex Molecule A(q) Associated with Autoimmune Arthritis.
(
- Contribution to journal › Article
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Mark
Rheumatoid arthritis: The role of reactive oxygen species in disease development and therapeutic strategies
(
- Contribution to journal › Scientific review
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Mark
Macrophages suppress T cell responses and arthritis development in mice by producing reactive oxygen species
(
- Contribution to journal › Article
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Mark
Nature's choice of genes controlling chronic inflammation.
2007) Symposium on Immunotherapy 2020 - Visions and Trends for Targeting Inflammatory Disease 4. p.1-15(
- Chapter in Book/Report/Conference proceeding › Paper in conference proceeding
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Mark
Lack of Reactive Oxygen Species Breaks T Cell Tolerance to Collagen Type II and Allows Development of Arthritis in Mice.
(
- Contribution to journal › Article
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Mark
The rheumatoid arthritis-associated autoantigen hnRNP-A2 (RA33) is a major stimulator of autoimmunity in rats with pristane-induced arthritis
(
- Contribution to journal › Article
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Mark
Arthritis suppression by NADPH activation operates through an interferon-beta pathway
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- Contribution to journal › Article
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Mark
Fragmentation of two quantitative trait loci controlling collagen-induced arthritis reveals a new set of interacting subloci
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- Contribution to journal › Article
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Mark
A case-control study of rheumatoid arthritis identifies an associated single nucleotide polymorphism in the NCF4 gene, supporting a role for the NADPH-oxidase complex in autoimmunity
(
- Contribution to journal › Article
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Mark
T cell responses to a non-glycosylated epitope predominate in type II collagen-immunised HLA-DRB1*0101 transgenic mice
(
- Contribution to journal › Article