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- 1999
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Mark
The Ala25-Thr mutation in the thrombomodulin gene is not frequent in Swedish patients suffering from ischemic heart disease [9]
(
- Contribution to journal › Letter
- 1998
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Mark
A common 4G allele in the promoter of the plasminogen activator inhibitor-1 (PAI-1) gene as a risk factor for pulmonary embolism and arterial thrombosis in hereditary protein S deficiency
(
- Contribution to journal › Article
-
Mark
The A20210 allele of the prothrombin gene is frequently associated with the factor V Arg 506 to Gln mutation but not with protein S deficiency in thrombophilic families [3]
(
- Contribution to journal › Letter
- 1997
-
Mark
Genetic and phenotypic analysis of a large (122-member) protein S-deficient kindred provides an explanation for the familial coexistence of type I and type III plasma phenotypes
(
- Contribution to journal › Article
-
Mark
Evaluation of original and modified APC-resistance tests in unselected outpatients with clinically suspected thrombosis and in healthy controls
(
- Contribution to journal › Article
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Mark
The factor VR5O6Q mutation causing APC resistance is highly prevalent amongst unselected outpatients with clinically suspected deep venous thrombosis
(
- Contribution to journal › Article
- 1996
-
Mark
Familial thrombophilia : clinical and molecular analysis of Swedish families with inherited resistance to activated protein C or protein S deficiency
1996) In Scandinavian journal of clinical and laboratory investigation. Supplementum 56(226). p.19-46(
- Contribution to journal › Scientific review
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Mark
Resistance to activated protein C, the FV:Q506 allele, and venous thrombosis
(
- Contribution to journal › Scientific review
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Mark
Inherited resistance to activated protein C caused by presence of the FV:Q506 allele as a basis of venous thrombosis
(
- Contribution to journal › Article
- 1995
-
Mark
Activated protein C resistance : from phenotype to genotype and clinical practice
(
- Contribution to journal › Article