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- 2008
-
Mark
Infectious Agents, the Contact System, and Innate Immunity
2008) 41st Congress of the German-Society-for-Transfusion-Medicine-and-Immunohematology/Interdiscip linary European-Society-for-Hemapheresis-and-Hemotherapy p.60-70(
- Chapter in Book/Report/Conference proceeding › Paper in conference proceeding
- 2007
-
Mark
M protein from Streptococcus pyogenes induces tissue factor expression and pro-coagulant activity in human monocytes
(
- Contribution to journal › Article
-
Mark
Thrombin activatable fibrinolysis inhibitor binds to Streptococcus pyogenes by interacting with collagen-like surface proteins A and B.
(
- Contribution to journal › Article
- 2004
-
Mark
M Protein, a Classical Bacterial Virulence Determinant, Forms Complexes with Fibrinogen that Induce Vascular Leakage
(
- Contribution to journal › Article
- 2003
-
Mark
The conversion of fibrinogen to fibrin at the surface of curliated E. coli bacteria leads to the generation of proinflammatory fibrinopeptides.
(
- Contribution to journal › Article
-
Mark
Contact activation by pathogenic bacteria: a virulence mechanism contributing to the pathophysiology of sepsis.
(
- Contribution to journal › Article
-
Mark
Modulation of membrane traffic by microorganisms.
(
- Contribution to journal › Article
- 2002
-
Mark
Identification of two protein-binding and functional regions of curli, a surface organelle and virulence determinant of Escherichia coli
(
- Contribution to journal › Article
- 2001
-
Mark
Staphylococcus aureus induces release of bradykinin in human plasma
(
- Contribution to journal › Article
- 2000
-
Mark
Severe lung lesions caused by Salmonella are prevented by inhibition of the contact system
(
- Contribution to journal › Article